Facial paralysis is usually acute in onset, leading to nerve function damage in the innervated area of the affected facial nerve, which mainly involves the facial expression muscles. Depending on the part of the facial nerve involved, the precursor symptoms of facial paralysis also have different manifestations. Peripheral facial paralysis is mostly caused by facial neuritis, and is often one-sided. Most of the patients tend to suddenly find that one side of the cheek does not move well and the corner of the mouth is skewed when they wash their face and gargle in the early morning. The main precursor symptoms are: headache on the sick side, pain behind the ear and neck, external sore throat, tinnitus, fatigue, etc., followed by paralysis of the facial expression muscles, frontal lines or disappearance, weak or incomplete closure of the eyes, eye fissure enlargement, drooping of the corners of the mouth, in the smile or teeth, drooping of the corners of the mouth and the facial skewing is more pronounced, the sick side can not be made to wrinkle the forehead, frowning, closing the eyes, puffing and pouting and other movements. When puffing the cheeks and whistling, the affected side of the mouth and lips can not be closed and air leakage; when eating, food residues are often retained in the sick side of the teeth and cheek gap, and often drool from the side; due to the tear point with the lower eyelid ectropion, so that the tear fluid can not be in accordance with the normal drainage and spillage. Depending on the site of facial nerve involvement, it may be accompanied by loss of taste in the anterior 2/3 of the tongue on the same side, auditory hypersensitivity, and impaired tear and saliva secretion. A few patients may experience discomfort in the lips and cheeks. In some patients, due to the reduced, delayed and incomplete eye closure, it may be secondary to ipsilateral corneal or conjunctival injury, with symptoms such as redness and swelling of the eyes. Central facial paralysis is mostly caused by cerebrovascular disease, which results in paralysis of the face below the eye fissure, with unaffected movements such as eye closure and frowning, which is distinctly different from peripheral facial paralysis, and the lesion sites and treatments are also very different. In conclusion, facial paralysis is relatively obvious and can usually be detected in time. If you find that you have facial paralysis, you must go to a regular hospital to clarify whether it is central facial paralysis or peripheral facial paralysis, and receive timely treatment.