What are the precursors of facial paralysis

Facial paralysis usually has an acute onset, leading to nerve function damage in the ipsilateral facial nerve innervation area, mainly involving the expression muscles in the ipsilateral face, and the symptoms of facial paralysis vary according to the different parts of the facial nerve involved. Facial paralysis caused by facial nerve palsy has an acute onset and often has no aura, but most patients have a history of exposure to cold and wind, and it mainly manifests as ipsilateral peripheral facial paralysis, which is mostly caused by facial neuritis and is often one-sided, and most patients tend to suddenly find that one side of their cheeks does not move well and the corners of their mouths are crooked when they wash their faces and gargle in the early morning. The main precursor symptoms are: shallow frontal lines on the sick side, weak eye closure, shallow nasolabial folds, etc. There may be paralysis of the facial expression muscles, disappearance of the forehead wrinkles, widening of the eye cracks, drooping of the corners of the mouth, drooping of the corners of the mouth and facial skewing is more obvious when smiling or showing teeth, and the sick side can not be made to wrinkle the forehead, furrowing of the brow, closing of the eyes, puffing and pouting of the mouth and other movements. When puffing the cheeks and whistling, the affected side of the mouth and lips can not be closed and air leakage; when eating, food residues are often retained in the sick side of the teeth and cheek gap, and often drool from the side; due to the tear point with the lower eyelid ectropion, so that the tear fluid can not be in accordance with the normal drainage and spillage. Depending on the site of facial nerve involvement, it may be accompanied by loss of taste in the anterior 2/3 of the tongue on the same side, auditory hypersensitivity, and impaired tear and saliva secretion. A few patients may experience discomfort in the lips and cheeks. In some patients, due to reduced or delayed eye closure and incomplete eye closure, it may be secondary to ipsilateral corneal or conjunctival injury, with symptoms such as redness and swelling of the eyes. Central facial paralysis is usually caused by cerebrovascular disease, and there are often cerebrovascular symptoms before the onset of the disease, such as dizziness and headache, vomiting, cognitive impairment, and trismus symptoms, and then facial symptoms begin to appear. To sum up, facial paralysis is more obvious and can be detected in time, but at the same time, we should pay attention to distinguish it from central facial paralysis caused by brain lesions. Central facial paralysis is paralysis of the face below the eye fissure, and movements such as closing the eyes and frowning are not affected, and there is a clear difference between the two, and the lesion sites and treatments are very different, so if you find that you are suffering from facial paralysis, you must go to a regular hospital to receive treatment.