Facial paralysis, also known as facial nerve palsy, is a paralysis of facial expression muscles caused by lesions of the facial nerve conduction pathway. Depending on the location of facial nerve damage, facial paralysis is clinically categorized into central facial paralysis and peripheral facial paralysis. In central facial paralysis, the damage is in the innervated area above the nucleus of the facial nerve, while in peripheral facial paralysis, the damage is below the nucleus of the facial nerve. Since the route of the facial nerve is adjacent to the anterior border of the transverse process of the atlantoaxial vertebrae after it exits the facial neural tube, the facial nerve can be stimulated or compressed in the event of misalignment of the upper cervical vertebrae or injury to the surrounding soft tissues, which can lead to dysfunction of the facial muscles of expression in the region of innervation. Peripheral facial paralysis caused by atlantoaxial joint deviation should be called “cervical facial paralysis”. This condition belongs to the categories of “tilting of the mouth and eyes”, “mouth fetish”, or “hanging thread wind” in Chinese medicine. “The occurrence of cervical facial paralysis has the following structural basis: 1. Facial nerve is the 7th pair of cerebral nerves, which are mixed nerves, the main component of which is motor fibers and is responsible for the movement of facial expression; the secondary component is the intermediate nerves, which contain afferent fibers of the body and viscera as well as efferent fibers of the viscera, and is mainly responsible for the secretion of the sense of taste and glands. Zhou Xuelong, Department of Bone and Joint Trauma, Hand and Spine Surgery, The First Affiliated Hospital of Guangxi University of Traditional Chinese Medicine (GXUTCM) 2, the facial nerve from the inner auricle to the mastoid foramen of the stem, this section is completely in the facial nerve canal, before leaving the facial nerve canal out of the mastoid foramen of the stem, but also divided into two branches, one for the Iwata nerve – leaving the facial nerve canal, in the pterygopalatine fossa for the visceral ganglion, joining to the trigeminal nerve, distribution to the eyes, After leaving the facial nerve canal, it changes into the visceral ganglion in the pterygopalatine fossa and joins the trigeminal nerve, which distributes to the eyes, nasal cavity, etc. and manages the secretion of the lacrimal gland and the mucous glands in the nasal cavity. Therefore, peripheral facial paralysis is divided into facial nerve canal injury and extra facial nerve canal injury according to the different parts of the injury. 3. The upper cervical spine has a close relationship with the upper cervical sympathetic ganglion, facial nerve, occipital nerve, occipital nerve and vertebral artery. Misalignment of the upper cervical vertebrae and the surrounding soft tissue injury can lead to the local blood circulation disorder and tissue metabolism abnormality, which in turn can lead to the organization of oedema, the formation of aseptic inflammation, and the stimulation of the neighboring facial nerves and lead to the dysfunction.