Gouty arthritis is caused by deposits of uric acid in the joint capsule, bursa, cartilage, bone and other tissues, resulting in lesions and inflammatory reactions. How to treat gouty arthritis? 1, the choice of uric acid-lowering drugs: 24-hour uric acid should be measured before the start of uric acid-lowering drug therapy to determine the patient’s uric acid metabolism and guide drug selection. For poor uric acid excretion type and most of the early and middle stages of gout, to promote uric acid excretion; for excessive uric acid synthesis type and most of the middle and late stages of gout, to inhibit uric acid synthesis. For severe gout stone gout, uric acid oxidase or a combination of drugs can be used. 2.Types of uric acid-lowering drugs: (1) drugs to promote uric acid excretion: benzbromarone, sodium bicarbonate, propoxur, etc.; (2) drugs to inhibit uric acid synthesis: allopurinol, febuxostat, etc.; (3) drugs to promote uric acid decomposition: uric acid oxidase (Precautions). 3. Preventive medication: (1) Drug anti-inflammatory prophylaxis: and should be continued if there is clinical evidence of persistent gout disease activity or if blood uric acid does not reach the standard. (2) Duration of treatment for attack prevention: 3 months after reaching the blood uric acid standard for those without gout stones and 6 months after reaching the blood uric acid standard for those with gout stones. (3) Treatment regimen for attack prevention: low-dose colchicine (0.5 mg qd or bid), low-dose NSAIDs, low-dose prednisone (≤10 mg qd) if both colchicine and NSAIDs are intolerant or contraindicated or ineffective.