Cirrhotic ascites is one of the most prominent clinical manifestations of the decompensated stage of cirrhosis, and there is no data on how long you can usually live after cirrhosis and ascites. The formation of ascites can be due to portal hypertension, causing an increase in hydrostatic pressure in the abdominal visceral vessels, leading to a decrease in tissue fluid reflux and then leakage into the abdominal cavity; due to a decrease in plasma colloid osmolality, leading to leakage of capillary fluids into the abdominal cavity; diminished hepatic inactivation of aldosterone and antidiuretic hormone, leading to secondary aldosterone and antidiuretic hormone increase, resulting in increased sodium and water reabsorption, decreased urine output and so on. Patients with cirrhotic liver ascites are recommended to seek timely medical treatment and standardize the treatment as prescribed by the doctor. Including the treatment of primary disease, diuresis, control of water and salt intake, correction of hypoproteinemia. Patients with large amounts of ascites can also be treated with puncture drainage and ascites concentration recovery to prolong survival.