The presence of patency in both fallopian tubes seriously endangers women’s health and often causes infertility and ectopic pregnancy. Some patients fail to pay attention to patency and later their condition worsens, leading to complete blockage, adhesions and fluid accumulation and eventual loss of tubal function. The causes of tubal incompetence or dysfunction are acute and chronic tubal inflammation. Tubal inflammation, which can be divided into tubal mucositis and peritubal inflammation, are both common causes of tubal pregnancy. In severe cases, tubal mucositis can cause complete blockage of the lumen and result in infertility. In mild cases, although the lumen is not completely blocked, adhesions in the mucosal folds can narrow the lumen or cilia defects can interfere with the normal operation of the fertilized egg in the fallopian tube and prevent it from implanting there. Peritubal inflammatory disease is mainly in the plasma membrane or plasma muscle layer of the fallopian tube, often resulting in peritubal adhesions, distortion of the fallopian tube, narrowing of the lumen, and weakening of peristalsis of the tube wall, which affects the movement of the fertilized egg. Tubal infections due to gonorrhea and Chlamydia trachomatis often involve the mucosa, while post-abortion or post-delivery infections often cause proximal blockage or patency of the fallopian tubes or perivasculitis. Tuberculous tubal infection is highly pathognomonic and causes infertility after cure, with occasional pregnancy, about 1/3 of which are tubal pregnancies. Tubulointerstitial isthmus is a special type of tubal inflammation. This lesion is due to diverticulosis of the mucosal epithelium of the fallopian tube into the isthmus and nodular hyperplasia of the muscle wall, which causes hypertrophy of the proximal muscle layer of the fallopian tube and affects its peristaltic function, resulting in obstruction of the fertilized egg and making it prone to tubal pregnancy. Inflammation causes deformation, narrowing, poor peristaltic ability, and even obstruction of the fallopian tubes, resulting in obstruction of the fertilized egg. Tubal infection can also be secondary to inflammation of organs or tissues surrounding the fallopian tubes, especially the formation of inflammatory adhesions around the umbrella of the fallopian tubes or the ovaries, causing partial obstruction of the umbrella of the fallopian tubes and even failure to draw the expelled oocytes into the fallopian tubes to meet the sperm leading to infertility. Therefore, patients who have suffered from adnexitis, septic appendicitis, tuberculous peritonitis, tuberculosis, endometriosis, fever, abdominal pain and puerperal infection after incomplete abortion, medication abortion, abortion, gonorrhea and other STDs, and patients with tubal malformation may all suffer from tubal incompetence. Therefore, the early diagnosis and treatment of tubal incompetence is particularly important. Through years of clinical analysis, I believe that selective tubal angiography (SSG) and interventional sparing procedure (FTR) under DSA machine are easy, safe, economical, effective, clear images, no incision, less painful for patients, less complications, and the preferred method for the diagnosis and treatment of tubal obstruction. It is the first choice for the diagnosis and treatment of tubal obstruction infertility.