Hepatic ascites, also known as cirrhotic ascites, is caused by cirrhosis of the liver leading to portal hypertension, abnormal liver function leading to a weakened ability of the liver to produce albumin to form hypoproteinemia, and a decrease in plasma colloid osmotic pressure leading to permeation of body fluids from intravascular to extravascular to form an ascitic fluid. Liver ascites is formed because of the loss of liver failure, so patients with liver ascites will first have the clinical manifestations of primary liver disease, hypoproteinemia patients in addition to ascites, but also can have lower limbs and other drooping parts of the body edema, when the amount of abdominal water is small often no obvious clinical manifestations or only show non-specific symptoms such as decreased appetite, a large amount of ascites can lead to abdominal distension, abdominal bulging, serious abdominal A large amount of ascites can lead to abdominal distension, abdominal swelling, and in severe cases, the abdomen is swollen like a frog’s belly, and the skin of the abdomen is taut and translucent. Treatment of hepatic ascites should first actively treat the primary disease, and at the same time can speed up the discharge of fluid from the abdominal cavity by using diuretic drugs, and can also use albumin to improve plasma colloid osmotic pressure, reduce body fluid exudation and reduce ascites. Severe massive ascites can be short-term small amount of ascites pumping, ascites autologous retransfusion and other new treatment means can also be used as appropriate.