There are significant geographic and racial differences in the incidence of prostate cancer, with the highest rates in the Caribbean and Scandinavia and the lowest rates in China, Japan, and former Soviet Union countries [1-9]. The incidence of prostate cancer among blacks in the United States is the highest in the world, and it has now surpassed lung cancer as the first tumor that endangers men’s health in the United States. The American Cancer Society estimates that there were approximately 230,110 new cases of prostate cancer in the United States in 2004, and 29,900 will die from this disease [10]. In Europe, approximately 2.6 million new cases of prostate cancer are diagnosed each year, and prostate cancer accounts for 11% of all male cancers and 9% of all male cancer deaths [11]. The incidence of prostate cancer in Asia is much lower than that in Europe and the United States, but has shown an increasing trend in recent years. In China, the incidence of prostate cancer was 1.71 per 100,000 population and the mortality rate was 1.2 per 100,000 population in 1993; the incidence increased to 2.0 per 100,000 population in 1997 [12] and to 4.55 per 100,000 male population in 2000. in 1979, there were only 98 new cases of prostate cancer in Taiwan, China; it had increased to 884 in 1995, with an age The standardized incidence rate reached 7.2 per 100,000 population, with 635 deaths and a mortality rate of 5.59 per 100,000 population in 2000. Patients with prostate cancer are predominantly older men, with a median age of 72 years for new diagnoses and a peak age of 75 to 79 years. In the United States, greater than 70% of prostate cancer patients are older than 65 years of age; it is rare in men younger than 50 years of age, but greater than 50 years of age, the incidence and mortality rates increase exponentially. The likelihood of prostate cancer is 0.005 % in individuals younger than 39 years, increasing to 2.2 % (1/45) in the age group 40-59 years and to 13.7 % (1/7) in the age group 60-79 years [12]. The risk factors that cause prostate cancer are not clearly defined, but some of them have been identified. One of the most important factors is genetic. If an immediate family member (brother or father) has prostate cancer, his or her own risk of developing prostate cancer increases by a factor of 1. The relative risk increases to 5-11 times when 2 or more immediate family members have prostate cancer [13-14]. Epidemiological studies have found that patients with a positive family history of prostate cancer are diagnosed approximately 6 to 7 years earlier than those without a family history [15]. A subpopulation (approximately 9%) of the population with prostate cancer is “true hereditary prostate cancer”, defined as three or more relatives with the disease or at least two with early onset (before age 55) [11]. Exogenous factors can influence the progression from so-called latent prostate cancer to clinical prostate cancer. The identification of these factors is still under discussion, but a diet high in animal fat is an important risk factor [16-18]. Other risk factors include low intake of vitamin E, selenium, lignans, and isoflavones. Sunlight exposure is negatively associated with prostate cancer incidence, and sunlight increases vitamin D levels, which may be a protective factor for prostate cancer [18]. In Asia, where prostate cancer is low, green tea consumption is relatively high and green tea may be a preventive factor for prostate cancer [19]. In conclusion, genetics is an important risk factor for the development of prostate cancer into the clinical type, and exogenous factors may have an important impact on this risk. The key issue now is that there is insufficient evidence to suggest that lifestyle changes (lowering animal fat intake and increasing intake of fruits, cereals, vegetables, and red wine) will reduce the risk of developing the disease [18, 20]. There are several studies that support these claims, and this information can be provided to the families of men with prostate cancer who come to ask about the effects of diet.