How does a brain attack patient get hiccups?

Hiccups, also known as eruptions, are a relatively common clinical symptom in patients with cerebral infarction, often affecting patients’ normal rest and sleep, and even affecting their appetite and causing them to have a psychological burden. The causes of hiccups in brain infarction patients are mainly the following: 1. Brainstem infarction: If a patient has brainstem infarction, frequent hiccups will occur. Because the ergodic center is located in the brainstem, if the brain infarction damages the ergodic center, it leads to the dysfunction of the ergodic center, and then the symptoms of hiccups can occur. In addition, if the brain infarct lesion stimulates the vagus nerve of the brainstem, it can also cause persistent hiccups; 2. Increased intracranial pressure: Some patients have increased intracranial pressure due to post-infarct edema, which stimulates the ergic center and can lead to hiccups; 3. Vagus nerve excitation: Brain infarct stimulation or mental tension can excite the vagus nerve and stimulate increased secretion of gastric acid, leading to stress gastric bleeding or stress gastric ulcers, leading to diaphragmatic hiccups. Gastric ulcer, resulting in spasm of the diaphragm, which can lead to hiccups; 4, gas: brain infarction patients because of restricted activities, the gastrointestinal tract activities are also reduced, easy to cause gas due to indigestion, will also cause hiccups; 5, lung infection: brain infarction patients due to sputum, respiratory function problems, often appear lung infection, if the inflammation involves the bottom of the lung, stimulating the diaphragm, can trigger hiccups; 6, other. For example, when brain infarction patients have neurological disorders and electrolyte disorders, the diaphragm appears to be stressfully elevated, and patients can show symptoms of hiccups.