Black crusts on the palate are commonly associated with pulmonary trichinosis. Pulmonary trichomycosis is an infectious disease of the lungs caused by pathogenic bacteria of the order Trichophyton. Although rare, it develops rapidly and has a high mortality rate. Clinically, Trichoderma and Rhizopus are more common, the former mainly invades the lungs, the latter mostly involves the sinuses, eyes, brain and gastrointestinal tract, and can be hematogenously disseminated to the whole body. Pulmonary trichinosis can be a primary infection, but also secondary to sinus lesions or trichinosis sepsis. The disease begins as a symptom of acute bronchitis and causes pulmonary solids and lung abscesses with signs of thrombosis and infarction when the lungs are involved. So what are the tests for black scorch on the palate? Here we will take a look. 1, the cause of pulmonary Trichophyton can be caused by inhalation of fungal spores in the air (3-6μm) or inhalation of fungal spores in the infected paranasal sinuses, or due to blood-borne dissemination of more distant lesions. Trichoderma is highly susceptible to invasion of the elastic lining of large and small blood vessels, causing thrombosis, hemorrhage and infarction. Clinical manifestations are nonspecific pneumonia, which can present with chest pain, dyspnea, hemoptysis, and no diagnostic significance on chest X-ray and routine bacteriological examination. It occurs more frequently in patients with leukemia and lymphoma than in patients with cancer, and primary pulmonary trichinosis can occur in diabetic patients. The prognosis is poor, with death in as little as 3 days and as long as 30 days. 2, nasal cerebral trichinosis acute, rapid and aggressive progress, manifested as facial pain, headache, drowsiness, serious cases can lead to blindness. Physical examination can be seen in the nose with brown, bloody slightly viscous secretions, the infected side of the palate with black crust. When the II, IV, and VI pairs of cerebral nerves are involved, it may also cause pupil dilatation, fixation, convexity, or ptosis. During disease progression, the fungus easily invades large blood vessels, causing infarction and necrosis in the brain with cerebral softening, and patients often progress from drowsiness to coma and die within 7 to 10 days. The morbidity and mortality rate of this type is 80% to 90%.