Hypoglycemia is a clinical phenomenon in which glucose concentration is lower than normal due to multiple etiologies and pathogenesis. The clinical manifestations of this disease are diverse, but mainly manifest as neurological symptoms such as sympathetic excitation and clinical syndrome of brain dysfunction. Clinically, it is generally divided into two categories: fasting hypoglycemia and postprandial hypoglycemia (reactive hypoglycemia). I. Diagnostic points 1. Clinical manifestations: the typical manifestation of hypoglycemia is Whipple’s triad. (1) Symptoms and signs of hypoglycemia: panic, palpitation, hunger, weakness, trembling of hands and feet, pale skin, sweating, increased heart rate, mildly elevated blood pressure, etc. Brain cell dysfunction due to insufficient supply of glucose and reduced oxygen supply; (2) Blood sugar lower than 2.5-2.8 mmol/L; (3) Rapid reduction or disappearance of symptoms after taking sugar or injecting glucose. Differential diagnosis: 1. Excessive secretion of insulin or insulin-like substances, such as insulinoma or extra-pancreatic tumor; 2. Antagonistic insulin hormone deficiency, such as hypopituitarism, hypoadrenocorticism, hypothyroidism; 3. Hepatogenic or nutrient supply deficiency; 4. Hypoglycemia caused by drugs. 1. Treatment of the cause: actively treat the original disease, such as removing insulinoma or eliminating the cause. 2.Treatment during the attack: lighter cases can be relieved by taking sugary food or sugary drinks orally; heavier cases can be relieved by injecting 50% GS 40-60ml sedately; in emergency cases, glucagon 1-5mg or dexamethasone 5-10mg or adrenocorticotropic hormone 25-50mg can be injected intramuscularly. Key points of nursing and rehabilitation After finding hypoglycemia, actively explain the nature of the disease to the patient, give analysis and comfort, and adjust the diet structure. V. Prevention points Actively treat the primary disease, eat less and more meals, and avoid starvation.