Atherosclerotic thrombotic cerebral infarction In addition to routine examinations such as blood and urine, attention should be paid to the examination of blood glucose, blood lipids, blood uric acid, blood viscosity, coagulation function, fibrinogen and electrocardiogram in order to find the causative factors for corresponding treatment. The imaging examination is preferred if available: MRI, because the lesion can be detected 2 hours after the onset of the disease, and the lesion in the brainstem and cerebellum can be clearly revealed, which provides the basis for hyperacute treatment. cT examination has no abnormal findings on the day of onset, especially within 6 hours, but hemorrhagic cerebrovascular disease can be excluded, and hyperacute treatment can be carried out without losing time. 24 to 48 hours later, the infarcted area can be detected Hypodense foci appear, but they are poorly demonstrated for brainstem infarcts. Cerebral angiography, including magnetic resonance angiography (MRA) and digital subtraction angiography (DSA), can show the site and extent of thrombosis and collateral circulation compensation. Transcranial Doppler ultrasonography (TCD) and local cerebral blood flow measurement can detect abnormalities and help in diagnosis. Physical and chemical examination of cerebral embolism MRI and CT have the same diagnostic value as cerebral thrombosis, which can not only clarify the nature of the disease, but also determine the site and extent of infarction as well as single or multiple foci. If high-density shadow, higher-density shadow or even isointensity shadow inconsistent with anatomical structures appear in the low-density area, it should be considered as hemorrhagic cerebral infarction, which has a higher incidence in cerebral embolism cases. In the case of fat embolism, fat globules are seen in cerebrospinal fluid, urine and sputum. Chest X-ray helps to understand the heart condition and the presence of infection and cancer in the lungs. Electrocardiography is routinely performed, and echocardiography can be done to further clarify the cardiac condition if necessary. When subacute bacterial endocarditis is suspected, changes in body temperature, blood picture, and blood sedimentation should be noted, and blood culture tests should be performed. MRA or DSA or TCD or even cerebral angiography should be performed when aortic arch macrovascular or cervical vascular lesions are suspected. Cerebrospinal fluid examination can be used when imaging equipment is not available, but caution should be exercised to prohibit it in patients with high intracranial pressure, as it may lead to brain herniation. If necessary, it should be performed with caution after strong cranial pressure-lowering measures have taken effect. The cerebrospinal fluid is mostly normal in patients with cerebral infarction, with only a few hemorrhagic infarcts in which red blood cells are seen and mostly after 24 hours of onset. In large infarcts, cerebrospinal fluid pressure is increased and the cell count and protein content may be slightly higher than normal several days after onset.