About half of anxiety patients clinically have nocturnal panic attacks, or acute anxiety attacks, and about 10% of anxiety patients have nocturnal attacks only. They are easily triggered by fatigue, tension, stress, serious life events, not sleeping well, illness and taking certain medications. Today, a patient from Shenzhen, who had a cold 3 months ago, took antipyretics before going to bed, woke up in the middle of the night with chest tightness, palpitations and dizziness, difficulty breathing, thought he was going to die, extremely nervous, fearful and scared, after many days were afraid to sleep at night, 24 hours to be accompanied by family members. There are also individual patients who have nightmares that induce acute anxiety attacks. Today, a pregnant woman was, for the first time, 8 months pregnant and woke up with a dream that she was carrying a cat, with a trembling heartbeat, cold sweat and panic (note: this case needs to be distinguished from a nightmare). During nocturnal anxiety attacks, symptoms of autonomic dysfunction such as choking sensation, dizziness, cold sweat and difficulty in breathing are more obvious, suggesting that autonomic nerves play an important role in nocturnal anxiety attacks. It is advisable to use antidepressants with better efficacy on plant nerve dysfunction and corresponding cognitive-behavioral training in treatment. Diet without alcohol, less coffee-containing beverages and a moderate low-salt diet is helpful. Talk about the formation mechanism of nocturnal anxiety attacks: The more typical clinical cases are those in which the patient wakes up from chest tightness, dyspnea or a feeling of suffocation, analyzed from the perspective of cognitive-behavioral medicine. 1. Increased return blood volume in the flat position, increased right ventricular excretion and increased pulmonary stasis. (Note: The mechanism of cardiopulmonary operation is such that the right ventricle delivers venous blood to the lungs, the blood is oxygenated in the lungs and then returns to the left ventricle, which pumps the oxygenated blood throughout the body and then returns to the right ventricle.) 2. The diaphragm is elevated in the horizontal position, compressing the lungs and making breathing more difficult than during the day. 3. At night, the vagus nerve tone increases, the heart rate decreases, the left ventricular blood output decreases, which aggravates pulmonary stasis, and at the same time, the vagus nerve excitation causes the smooth muscle of the trachea to contract, the diameter of the tube shrinks and the resistance increases, which increases the burden of breathing. The above is normal physiological nighttime dyspnea, if there is no serious cardiopulmonary disease, is usually unhindered. 4, anxiety patients are often insecure and subconsciously have a serious fear of death, resulting in extreme sensitivity to changes in the body, especially to such life-threatening physiological activities as heartbeat and breathing. The above nighttime breathing difficulties are usually not felt by normal people, but can be detected by anxious patients, thus waking them up or waking them up. 5. After waking up, the negative cognition of the anxious patient interprets the physical symptoms as a serious physical illness, which leads to more nervousness and anxiety, so that the anxiety, negative cognition and physical symptoms aggravate each other in a vicious circle, which is magnified step by step and rapidly leads to anxiety attacks.