Topical corticosteroids (hormones for short) are commonly used in dermatology clinics and are important therapeutic means. Due to the lack of understanding of the therapeutic effects and adverse reactions of topical hormones, the inability to grasp the indications and contraindications, coupled with the abuse of medical origin or non-medical misuse, resulting in long-term use of the skin caused by localized adverse reactions, especially after discontinuation, resulting in the recurrence of the original inflammation or rebound, have to continue to use topical hormones to inhibit inflammatory reactions, the formation of hormone-dependence, which puts forward the “hormone-dependent dermatitis (steroids-dependent dermatitis)”. “Hormone-dependent dermatitis (steroids-dependent dermatitis)” diagnostic name, and in the clinical widely accepted by doctors and applied to the diagnosis of patients. Domestic even formulated an expert consensus on related diseases to standardize the understanding of this disease, but its definition and diagnostic criteria lack unity. It is doubtful whether the diagnostic name of hormone-dependent dermatitis has been abused and whether it is a separate disease. With hormone-dependent dermatitis or hormone withdrawal dermatitis to check the relevant literature database, it is not difficult to find Chinese articles to be significantly more than foreign literature, this difference in addition to the existence of a certain amount of abuse of topical hormones in China or unregulated use of the relevant, may be to a large extent there is a cognitive error. Check the definition of hormone dependence or addictive dermatitis, the real meaning is mainly refers to the long-term use of hormones caused by a special inflammatory conditions, mainly occurring in the face, also known as facial red face syndrome (red face syndrome) or erythema burnig skin syndrome (red burnig skin syndrome), but also can occur in the scrotum, caused by red scrotum syndrome (red scrotum syndrome), can also occur in the scrotum. It can also occur in the scrotum, causing red scrotum syndrome. The disease is mainly seen in patients with atopic dermatitis, which accounts for 90% of cases, but can also occur in other diseases, including seborrheic dermatitis and dry dermatitis. From the manifestation of the disease, local skin lesions in addition to erythema and papules and other inflammatory reactions, often accompanied by capillary dilatation and mild atrophy of the skin, which are very similar to the topical hormone residual local adverse reactions. Patients’ self-conscious symptoms mainly include facial tightness, burning sensation, dryness, and a considerable number of patients lack itching, or non-major symptoms, which is somewhat different from general dermatitis. Patients mostly complain of paroxysmal flushing, and become the precursor of each recurrence. The site of onset is most often seen on the face, other parts of the body even if long-term topical hormone rarely occurs, the reaction of this condition occurs under certain specific conditions, may be closely related to the rich vascular structure of the face, which is similar to the mechanism of the occurrence of rosacea. As this group of patients is dominated by facial erythema, capillary dilatation, episodic flushing, and can be triggered or aggravated by minor stimuli such as changes in indoor ambient temperature, eating stimulating foods, and emotional excitement, the appearance of pimples or pustules suggests the presence of localized vascular hyperreactivity. Local examination of the lesions may reveal an increase in the number of microorganisms such as Malassezia furfur colonizing the area, as well as an increase in the number of helminths. Therefore, no matter its pathogenic triggers, clinical manifestation forms, or the process of flare-ups, to a large extent, it is in line with the characteristics of rosacea, especially with the erythematous capillary dilatation type and papulopustular pustular type of rosacea, which are more similar. In foreign monographs, the abuse of topical hormone preparations is also mentioned as a factor when describing the causes or triggers of rosacea, and even the name “corticosteroid-induced rosacea” is proposed as a special type of rosacea. The name of this disease clearly emphasizes dependence or addiction, but most of the patients encountered in the clinic have already stopped using hormones, and it is not a manifestation of the need to repeat the use of hormones in order to control the disease. Clinically, once there is a history of topical hormone use, facial manifestations of erythema, papules, pustules and capillary dilatation, the diagnosis of hormone-dependent dermatitis, obviously there is a misuse of this diagnostic name may be. Therefore, hormone-dependent dermatitis is not a common stand-alone disease, and is seldom hormone-dependent dermatitis in the true sense of the word, but more often belongs to the category of rosacea or hormone-induced acne-like rashes, except that topical hormone use may be one of the causative factors. If we agree to view hormone-dependent dermatitis more as a type of rosacea or acneiform rash, then treatment should be directed at the pathophysiology of rosacea or acneiform rash. Firstly, stop using all hormonal preparations, including compounded herbal preparations or blemish removers that may contain hormones; secondly, the pathogenesis of the disease is based on localized vascular hyperreactivity, avoiding various factors that trigger or exacerbate vascular reactions, such as reducing the intake of strong tea, coffee, and spicy foods, avoiding staying in a warm and airtight indoor environment, and maintaining emotional stability; inflammatory reactions mediated by a variety of factors are considered to be an important basis of pathogenesis The increase in the number of helminths and Malassezia furfur at the site of the lesion is a secondary phenomenon, or even considered a concomitant phenomenon, and therefore anti-inflammatory therapy is more important than anti-microbial therapy; the patient’s feeling of tightness and dryness of the skin suggests that the skin barrier is impaired, and therefore the use of measures to help restore the skin barrier, such as moisturizing skin care agents, is an important basic treatment. In the absence of contraindications, oral tetracyclines such as doxycycline and minocycline are an appropriate choice, as they have a clear anti-inflammatory effect and are also anti-capillary proliferative. The pathogenesis of this disease is based on local vascular hyperreactivity, removal of local dilated capillaries is an important condition for the prevention of recurrence, the choice of 585 dye laser, intense pulsed light (IPL) to carry out the treatment in a prudent and orderly manner may receive a certain effect, but pay attention to the choice of the appropriate timing of the treatment, the treatment may be aggravated by local inflammatory reaction, left behind by the hyperpigmentation, hypo-pigmentation, and other adverse reactions, need to be fully communicated with the patient. patients to fully communicate. At the same time need to treat the face of the original disease such as seborrheic dermatitis, photosensitive dermatitis, etc., is equally important to control the condition. In conclusion, hormone-dependent dermatitis is not a common independent disease, and its meaning should be strictly defined to prevent the misuse of this diagnostic name. After the topical use of hormones, facial erythema, capillary dilatation, inflammatory papules and other patients, more should be diagnosed as rosacea or acne-like rash, not to diagnose hormone-dependent dermatitis. Treatment is based on elimination of the causative agent and reduction of local vascular hyperreactivity, with topical or systemic anti-inflammatory therapy as an important tool, and lasers or skincare products to help restore the skin barrier as important measures.