Typical chronic inflammatory demyelinating radiculopathy (CIDP) presents as a chronic, more or less symmetrical, sensorimotor neuropathy with loss of tendon reflexes and sensory symptoms in 85% of patients, of which 6% show pure sensory nerve involvement. The majority of patients have motor nerve involvement, of which 22% have only motor nerve involvement. In typical cases, both the distal and proximal extremities are involved, especially in the lower extremities, which may be related to nerve root involvement. Sensory nerve involvement is relatively milder than motor nerve involvement, and large fiber involvement is more severe than small fiber involvement. 3 large case studies found respiratory muscle involvement in 15% of patients, ball palsy in 6-10%, facial muscle palsy in 6-15%, and oculomotor nerve involvement in 4%. Autonomic nerves are infrequently involved compared to GBS. The clinical presentation is varied, and patients may have predominantly cranial nerve palsy, which may even be the first symptom. About 20% of patients present with painful sensory abnormalities. About 3% of patients develop postural tremor, which may even lead to disability. Muscle atrophy and contractures may develop as the disease progresses. Bowed feet and flogging toes may be seen in individual patients who have had the disease since childhood. Peripheral nerve hypertrophy is seen in CIDP, but more commonly in hereditary demyelinating neuropathies. 1-7% of patients may have optic papillar edema, which may be associated with elevated cerebrospinal fluid proteins.