Because this procedure is relatively new to neurologists, surgeons, and ophthalmologists nationwide, and many patients come to the clinic with questions and doubts, I have decided to publish this section here. This will help more doctors understand the procedure, which will also help patients and families understand the procedure and help communication and understanding between doctors and patients. Anesthesia accidents (very low chance), there are risks with any anesthesia; they are covered by the entire surgeon’s surgical team; if they occur, they are fully resuscitated. Drug allergic reaction, or rare poisoning reaction (the chance is very low); any drug may have side effects on the body, ranging from nausea, vomiting, rash to anaphylactic shock and death; the entire surgeon’s surgical team will guarantee this; if it occurs, all efforts will be made to save the patient. Postoperative infection, any surgery has the possibility of potential infection, orbital infection may occur in serious cases, or even spread to the skull (the chance of occurrence is very low); preventive measures: strict intraoperative aseptic operation, perioperative anti-infection; if it occurs, all-out anti-infection. Intraoperative retrobulbar hemorrhage, which usually occurs when the ciliary vessels are damaged, has a low chance of occurrence; countermeasures: reducing the pull on the eye is sufficient; intraoperative damage to adjacent tissue structures, such as the ciliary segmental nerve or the long ciliary nerve or other orbital lid nerves, causes irregular pupils or dilatation; this complication has a 10% chance of occurrence as reported abroad. We had a case in the early stage. Because of the disconnection of the medial rectus muscle after surgery, although the medial rectus muscle is reset after surgery, it cannot be guaranteed to be properly reset, so mild internal or external strabismus may occur in the operated eye after surgery, and some patients have diplopia (both eyes); one case occurred and was observed for two months to improve; or wearing a trigeminal lens can resolve it. For patients with early optic nerve compression, about 80% of vision can be reversed; for patients with late compression, 40-50% of vision can be Qin all reversed, but the vast majority of residual vision is preserved. In some patients whose intracranial pressure is not effectively controlled and whose optic nerve has been compressed for a long time, the postoperative visual acuity does not improve, improves (generally very poor visual acuity at manual or light-sensitive level), or remains invisible or (no preoperative light-sensitive); the occurrence of such undesirable results depends mainly on the degree of preoperative optic nerve injury, the degree of compression, the time of compression and the height of intracranial pressure. There have been reports from abroad of postoperative retrobulbar hemorrhage that compressed the optic nerve, requiring an exophthalmos or reoperation to access the retrobulbar area to stop the hemorrhage, which has not occurred; there have been reports from abroad of complete loss of vision after surgery, which has a very low chance of occurring due to excessive pulling on the eye or compression by retrobulbar hemorrhage, which has not occurred; in addition to the above, there are other complications that may occur with this medical measure. In general, we have successfully done more than 100 cases and saved many patients; in terms of more than 3 years, there has not been a single patient death, no case of postoperative blindness due to surgery, and no case of infection; in my personal opinion, this procedure is a relatively high safety factor compared to other procedures (ventriculoperitoneal drainage, lumbar pool peritoneal drainage, etc.).