Cerebral hemorrhage, commonly known as cerebral hemorrhage, refers to bleeding in the brain parenchyma. Although cerebral hemorrhage can be caused by trauma, in the classification of acute cerebrovascular disease, it generally refers to non-traumatic, also known as primary or spontaneous cerebral hemorrhage. Although cerebral hemorrhage can originate from arteries, veins, and capillaries in the brain and can be caused by a variety of reasons, bleeding from small arteries in the deep brain is the most common, and hypertension is the most common cause, the latter condition being called hypertensive cerebral hemorrhage. Hypertensive cerebral hemorrhage is seen in patients with long-term hypertension, when blood pressure rises further abruptly and cerebral blood vessels suddenly rupture and bleed. Occasional increases in blood pressure, which can occur in everyone, rarely lead to cerebral hemorrhage. This is because the occurrence of cerebral hemorrhage depends not only on changes in blood pressure, but is also closely related to the health of the cerebral vessels. Patients with chronic hypertension are particularly susceptible to lesions in the small cerebral arteries, where the arterial walls become degenerated, necrotic, or form microaneurysms. When blood pressure rises sharply, it causes these weak parts to rupture and bleed. Cerebral hemorrhage is often sudden, usually occurring during the day when the patient is emotionally excited, overexerted, or highly stressed; some occur after drinking alcohol or during bowel movements. A small number of patients have the onset during sleep, especially during nightmares, or during sexual intercourse. At the beginning of the disease, most patients feel sudden headache or dizziness, and then the mouth and eyes are distorted, half of the body is weak, and heavy patients soon become confused or even comatose. At this time, the patient often breathes deeply, snores loudly, has a generally rosy complexion, a slow and strong pulse, very high blood pressure, and frequent vomiting. Paroxysmal tonicity of the limbs may occur, and finally the breathing becomes irregular; some patients have a sharp rise in body temperature or vomit coffee-colored liquid, which are all signs of poor prognosis.