Eyelid skin laxity syndrome: also known as progressive lid laxity, is a rare eyelid disorder characterized by recurrent eyelid edema in adolescents, with thinning of the eyelid skin, loss of elasticity, increased wrinkles, and color changes, which can be complicated by clinical manifestations such as lacrimal gland prolapse, ptosis, and shortening of the transverse lid fissure. It is mostly seen in young women, and the pathogenesis is unclear. Klauder classifies it into 4 types 1. Fuchs type; recurrent episodes of eyelid water causing skin laxity; 2 adult type: occurs in middle age; 3. genetic type: family history; 4. non-above mentioned types. The hypertrophic type is mainly due to orbital septal dysplasia, with fat herniation caused by repeated inflammatory stimuli, and is characterized by fullness and hypertrophy of the upper eyelid, mostly accompanied by prolapse of the lacrimal gland; the atrophic type is due to long-term chronic inflammatory stimuli resulting in soft tissue atrophy and thin and loose skin lesions, mainly characterized by sunken upper eyelids and increased wrinkles. Treatment for lid laxity is primarily surgical and the timing of surgery is usually when the lesion is in a stable or quiescent phase. Surgery: A blepharoplasty incision is used to correct the laxity of the skin and reposition the tear duct. Or lid skin laxity and ptosis.