How to effectively prevent the progressive increase of serum alkaline phosphatase

In liver metastases or liver disease from cancer, serum alkaline phosphatase may be progressively increased, often exceeding 20 gold A units, and γ-glutamyl transpeptidase (γ-GT) may be positive. Glutamate aminotransferase and other liver function changes are not significant. Patients tend to die within a short period of time. How to effectively prevent serum alkaline phosphatase can be progressively increased? Protein is synthesized in the liver 11-14 grams per day. The protein in the plasma will reflect the number of cells in the liver function, and a sustained decrease below 3 grams is noted in opposition, which can have a significant impact on the disease. The oxidation of synthetic fatty acids and the generation of carcasses are done in the liver. Increased alkaline phosphatase is seen in liver metastasis of cancer or liver disease with jaundice acute and chronic hepatitis, cirrhosis, bile duct obstruction, liver cancer, etc. Nutrition is the best substance to maintain liver function. Nutritional balance is an important factor that leads to good or bad liver function, and also has a particularly high nutritional requirement and is the basis for maintaining normal structure and function of the liver. Too much or too little nutrition can cause abnormal liver function. When nutrition is poor, liver cells are damaged and metabolism is impaired, such as the metabolism of proteins, fats and vitamins and minerals. Protein is synthesized in the liver 11-14 grams per day, the protein in the plasma will reflect the number of cells in the liver function, consistently below 3 grams is noted to be antagonistic, the impact on the disease will be great, the oxidation of synthetic fatty acids and the generation of carcasses are done in the liver. Excessive fat accumulation is called fatty liver, caused by excess liver fat and phospholipid deficiency. Carbohydrates are hydrolyzed by enzymes into glucose, which is absorbed by the intestinal mucosa and passes through the meridians to the liver, where part of it remains for metabolism and part passes through the liver to the blood. In the liver also causes glycogen called hepatic glycogen, which can be temporarily stored in the liver, oxidized for the needs of the liver, into fatty acids and amino acids, etc., and then decomposed into glucose, into the blood, when the stored sugar is reduced some amino acids, glycerol, lactic acid industry can synthesize glycogen, which is called the hepatic glycogen xenobiotic effect. Vitamins are involved in metabolism, and poor liver function can also cause vitamin deficiency. Conditions that trigger liver disease are nutritional factors, diet, infection and metabolic abnormalities caused by genetic deficiencies, such as hepatitis, cirrhosis, liver ascites, liver fat, etc.