In patients with cerebral infarction, numbness all the way to the right side is mainly due to infarction of the left internal capsule, thalamus, and brainstem, which causes damage to the right spinal thalamocortical tract or thalamocortical tract. Internal capsule infarction is characterized by loss or diminution of hemianesthesia on the side contralateral to the lesion, often accompanied by hemianopsia as well as hemiparesis. Thalamic infarcts may present with persistent numbness on the side contralateral to the lesion, which is more severe distally than proximally, and is often accompanied by spontaneous pain in the affected limb called thalamic pain. On the other hand, brainstem infarcts are often characterized by cross-cutting sensory deficits, with patients experiencing paraplegia on the opposite side of the lesion, as well as facial numbness and loss of nociception on the same side of the lesion. Treatment is mainly to give aspirin anti-platelet aggregation, as well as statin lipid regulation, plaque stabilization therapy, and should actively control risk factors. Common risk factors for cerebral infarction include hypertension, hyperglycemia, hyperlipidemia, hyperhomocysteinemia, as well as obesity, too little exercise, and smoking. Patients should eat more fresh fruits and vegetables, actively participate in sports, and ensure quality sleep.