In general, diabetic foot refers to infection, ulcer formation and/or necrosis of the lower extremity in diabetic patients due to combined neuropathy and various degrees of vasculopathy. The diabetic foot is not a generalized concept, but is divided into three types. Each type has a different treatment plan for diabetic foot, and if confused, it can easily lead to delay and serious consequences. The first type is the neuropathic type. This type of foot disease is clinically manifested by numbness and loss of sensation in the foot. Diabetic neuropathy can involve sensory nerves, motor nerves and autonomic nerves, and sensory neuropathy is mostly a glove-like distribution of sensory abnormalities or even sensory loss, so that the patient’s sense of protection against temperature, pain, pressure, etc. is weakened or lost, and under the action of external factors such as burns, foreign bodies, trauma, etc., foot ulcers will occur in the foot without protection. The second type is the arterial ischemic type. This type of diabetic foot is extremely easy to be misdiagnosed and leads to the most serious consequences. Clinically, due to the long-term impact of high blood sugar in diabetic patients, the blood vessels in the lower extremities are hardened, the walls of the blood vessels are thickened, and the elasticity is reduced, so that blood clots can easily form and collect into plaques, resulting in occlusion of the blood vessels in the lower extremities, thus causing tissue lesions in the lower extremities. The “foot” is the farthest from the heart, and the ischemia is the most serious, resulting in edema, blackening, decay, necrosis, and gangrene. The third type is the mixed type. This means that neuropathy and arterial ischemia are present at the same time.