Facial palsy is not just a disease, but often a symptom of other diseases that manifest themselves. The facial nerve has the ability to repair itself after a lesion occurs, but the ability to heal itself varies significantly depending on each person’s constitution, as well as the cause and degree of the disease, and reasonable treatment should be given early. The most common form of facial palsy is idiopathic facial nerve palsy, which is characterized by peripheral facial palsy, paralysis of facial expression muscles on one side, loss of frontal lines, inability to frown, incomplete eyelid closure, shallow nasolabial folds, drooping corners of the mouth, drooling, cheek puffing, whistling and air leakage. In the acute stage, oral hormone therapy combined with B vitamins should be used, and if necessary, antiviral drugs such as acyclovir can be added, and physical therapy such as ultrashort wave heat therapy, infrared irradiation or local hot compresses can be performed near the stem mammary foramen, while attention should be paid to protecting the cornea. It is beneficial to prevent the emergence of sequelae. The factors affecting the recovery of facial neuritis depend mainly on the severity of the disease and whether the treatment is timely and reasonable. The prognosis is better for young patients with mastoid pain at onset, those with diabetes, hypertension, arteriosclerosis, angina pectoris or previous history of myocardial infarction, and older patients with a poor prognosis, and the recovery rate for milder facial palsy can reach more than 92% regardless of treatment. Usually, about 80% of patients can recover within a few weeks or 1 to 2 months, and the recovery of taste within 1 week suggests a good prognosis. Incomplete facial palsy can recover or be cured within 1 to 2 months, while complete facial palsy takes 2 to 8 months or even 1 year to recover and often leaves after-effects. Another common cause of facial palsy is cerebrovascular disease, which is manifested as central facial palsy, manifesting as paralysis below the facial eye fissure, and movements such as eye closure and frowning are not affected. Unlike peripheral facial palsy, central facial palsy is damaged above the facial nerve nucleus to the cerebral cortex, and usually does not heal on its own, and should be treated promptly for the original brain disease, and through early and reasonable treatment and later rehabilitation exercises, no sequelae can be left. In conclusion, although the facial nerve has a certain ability to heal itself, early treatment should be carried out to clarify the cause of the occurrence of facial palsy and promote the recovery of nerve function so as to reduce complications and sequelae.