Diabetic wounds have been bad, in fact, this is what we often refer to as chronic ulcer wounds, referred to as slow trauma, the wound has been stagnant at a certain stage can no longer continue to move forward, or even worsen in a worse direction. Here, we will first take you through the possible causes. In most cases because of local necrotic tissue in the wound, foreign bodies, and the presence of bacteria producing metalloproteinases – which destroy the basis of tissue building (chemokines, growth factors, and mitosis); bacteria producing destructive enzymes – which consume local oxygen, nutrients. Bacteria form bacterial biofilms, making infection difficult to control; cells at the wound margin begin to age or have reduced regenerative capacity. In addition to the characteristics of chronic ulcers, diabetic foot ulcers also have hyperglycemic factors, local neuropathy leading to loss of protective nociception and foot deformity, vascular lesions leading to severe limb ischemia and infection leading to osteomyelitis and local gangrene of the foot. I have always emphasized that the same timing of wound debridement applies to diabetic foot ulcer debridement, and it is important to emphasize that ischemia and infection affect the choice of debridement timing, with immediate debridement of ischemic limbs with wet gangrene or abscess formation; dry gangrene without cellulitis awaiting treatment after improving local circulation. Regarding the specific implementation of debridement, the whole process is extremely complex, for example, regarding the skin tissue, the border between necrotic and normal tissue is clear and debridement starts from the demarcation; the border between necrotic and normal tissue is unclear, starting from the center of the necrotic tissue, fully draining the pus and gradually cleaning from the central skin to the edges one by one; small embolized veins are visible in the skin at the wound edge suggesting interruption of local microcirculation and the need to expand the debridement The scope needs to be expanded. For subcutaneous tissue, fascia, tendon, muscle and bone debridement, normal and abnormal fat, fascia, tendon, muscle and bone are identified. After removal of necrotic tissue, normal fat, fascia, tendon and bone are kept moist to maintain activity and avoid dry necrosis. To ensure complete removal of the necrotic tendon, the distal end of the necrotic tendon is removed whenever possible. In conclusion, it is recommended that you should prefer a specialized department for treatment, in which case the doctor is more experienced, has a better ability to identify the trauma, and a good debridement will significantly improve the overall treatment effect. On the contrary, if these are not done well, the treatment is just not effective, and it is even possible to develop in a serious direction.