Cerebral amyloid angiopathy is the leading cause of spontaneous cortical and subcortical hemorrhage in normotensive elderly people, preferably in the frontal and frontotemporal lobes, characterized by a wide distribution of outer and middle membrane β-amyloid in small and medium-sized vessels in the cortex, subcortex and soft meninges. Clinical manifestations are neurological deficits (stroke), TIA oxygen symptoms or dementia. Cerebral amyloid angiopathy causes 2% of all types of cerebral hemorrhage but accounts for 38%-74% of elderly patients with non-traumatic cerebral hemorrhage with normal blood pressure. Therefore, cerebral amyloid angiopathy is an important type of cerebral hemorrhage. Guo Dazhi, Department of Hyperbaric Oxygenation, Naval General Hospital As a therapy, the main treatment mechanism of hyperbaric oxygen therapy is to improve tissue ischemia and hypoxia, in addition to a variety of functions such as reducing cerebral edema and improving brain metabolism. In China, recovery from cerebral hemorrhage is a non-emergency indication for hyperbaric oxygen therapy, and many small-sample clinical studies have shown that hyperbaric oxygen therapy can reduce neurological deficit scores and improve efficiency in patients with cerebral hemorrhage. Therefore, the author believes that hyperbaric oxygen therapy is beneficial for patients with cerebral hemorrhage including cerebral amyloid angiopathy cerebral hemorrhage. The degree of improvement of neurological deficits depends on the patient’s age, the degree and location of cerebral hemorrhage, the presence or absence of brain herniation, complications, and the duration of cerebral hemorrhage sequelae, etc., which varies from person to person.