Hard edema of the palm is a localized edema of the palm tissues due to inflammation from trauma, infection, autoimmune factors, etc. For various reasons, some edema can persist for more than a month to 1-2 years. Sclerosing edema appears in the uncinate of the palm, with early flushing and a characteristic large patchy peeling of the fingers during the recovery period, commonly at the skin junction of the nail bed. The causes of hard edema of the palm are lymphoid primary lymphoedema (congenital lymphoedema early onset lymphoedema), secondary lymphoedema (tumor infection surgical radiation, etc.); dengue, boils, snake venom, poisoning, etc. Allergic urticaria, serum sickness and allergic reactions to food and drug irritation, topical drugs, etc. Vascular neurogenic can be allergic reactions or neurogenic can be induced by insect mechanical stimulation warm heat stimulation or emotional excitement; some cases are genetically related; trauma, stress, etc.; Kawasaki disease. Venous obstructive tumor compression or tumor metastasis, local inflammatory venous thrombosis, thrombophlebitis, scar contraction, and trauma. It can be divided into chronic venous insufficiency superior vena cava obstruction syndrome, inferior vena cava obstruction syndrome, and other venous obstruction. For various reasons, some edema may persist for more than a month to 1-2 years, and we call it “persistent edema” if the edema persists for more than 6 weeks after the injury and does not tend to subside, “in some of these cases the edema fluid is rich in protein and collects a large amount of fibrin, which together with the inflammatory proliferation In some of these cases, the edema fluid is rich in protein and will collect a large amount of fibrin, which will react with the inflammatory proliferation to form scar tissue and harden the skin and subcutis. This results in the formation of “hard edema.”