Acute cholecystitis and chronic cholecystitis are the result of bacterial attack on the gallbladder, edema and inflammation of the gallbladder wall, resulting in impaired blood supply to the wall and further inflammation, increased absorption of bile acids and lecithin by the gallbladder, and imbalance of the normal ratio of bile acids. Bacterial infection can make bile easily become acidic and cholesterol easily precipitated. The mucous membrane of the cyst wall is shed due to inflammation of the gallbladder. Inflammation of the gallbladder and bile ducts can cause stagnation of bile, and these are important factors in the formation of stones. If stones are formed, due to the stimulation of stones or stones embedded in the gallbladder neck or gallbladder duct, poor bile excretion, chemical stimulation of the gallbladder wall by bile, and very easy to be infected by bacteria, acute cholecystitis occurs, or make acute attacks of chronic cholecystitis. In patients with cholecystitis, more than 90% are caused by stones and 10% are non-stone cholecystitis. There is also chemical cholecystitis, due to pancreatic reflux into the gallbladder, with active pancreatic enzymes can cause acute cholecystitis. In some dehydrated patients, acute cholecystitis can also occur due to increased concentration of bile salts. Bacterial cholecystitis, such as sepsis, tuberculosis, typhoid fever, radiation bacterium and other bacterial infections causing hematogenous dissemination and bacterial cholecystitis. It can also occur as a result of trauma, surgery causing insufficient blood volume, vasospasm, slow blood flow, and thrombosis of the gallbladder artery resulting in ischemic necrosis of the gallbladder wall and secondary gallbladder infection. Therefore, cholecystitis is closely related to cholelithiasis and must be prevented and treated simultaneously.