Allergic rhinitis is an IgE-mediated type I allergic disease, which is a common and frequent disease in otolaryngology worldwide. Epidemiological studies have reported that the prevalence of AR is 10%-40% in foreign countries and 37.74% in China, with about 20 million people suffering from the disease each year. With the progress of industrialization, modern lifestyles and rapid changes in human ecology, the prevalence of AR has a global trend of growth. Although AR is not a major disease, it has a great impact on patients’ health and quality of life, daily activities and social intercourse, and the resulting complications (such as sinusitis, nasal polyps, otitis media, asthma, etc.) add to the problem, so AR is not only a medical problem, but also a social problem. Deviated nasal septum leads to abnormal anatomical structures in the nasal cavity and causes mechanical obstruction. Long-term unbalanced stimulation of both nasal cavities produces associated clinical symptoms through nasal sensory-parasympathetic reflexes. Abnormal nasal structures, such as nasal septum crista, are susceptible to overstimulation by abnormal airflow, causing abnormal neural reflexes and nerve dysfunction in the nasal mucosa, releasing more chemical mediators, such as vasoactive intestinal peptide (VIP) and substance P (SP), producing symptoms such as nasal itching, sneezing and nasal congestion. At the same time, the nasal mucosa is stimulated by the craggy or spiny protrusions of the nasal septum, which stimulate the nasal sensory nerve endings and induce abnormal sensory reflexes; abnormal airflow in the nasal cavity increases the chance of mucosal contact with allergens; nasal obstruction affects the arrival of local medication at the site of action, etc. Chemical mediators produced during the pathological process of allergic rhinitis, such as alkaline eggs (MBP) and cationic eggs (ECP) released by eosinophils, can damage the epithelium of the nasal mucosa, leaving its sensory nerve endings in a hypersensitive state and lower than normal sensory thresholds. Therefore, deviated nasal septum is closely related to the development of allergic rhinitis and is an important factor in inducing allergic rhinitis. After surgical correction of the deviated septum, the mechanical obstruction of the nasal cavity is relieved, nasal ventilation is improved, and the therapeutic effect is achieved by removing the undesirable irritation. Studies have confirmed that in the pathogenesis of allergic rhinitis, the nerves that regulate the vasodilation and glandular secretion of the nasal mucosa are the anterior septal nerve and the parasympathetic part of the pterygoid nerve. The anterior septal nerve can be divided into the septal branch and the lateral nasal branch, which are both sensory nerves and contain more parasympathetic fibers, and can regulate the vasodilation of the nasal mucosa and the secretion of the glands. The nasal septal branch of the anterior septal nerve is mainly distributed in the upper anterior part of the nasal septum and enters the vertical plate of the sieve bone, innervating the plasma glands in the upper part of the nasal septum. When the nasal mucosa is exposed to allergens, the sensory nerve endings of the nasal cavity on both sides are stimulated and the parasympathetic nerve releases acetylcholine locally, causing enhanced secretion of the glands and provoking clinical symptoms such as nasal itching, sneezing and runny nose. The septal deviation correction surgery destroys the nasal septal branch of the anterior sieve nerve, cuts off the abnormal nerve reflex and reduces the release of acetylcholine, thus relieving the symptoms of allergic rhinitis. At the same time, the extensive stripping of the mucous cartilage membrane and mucoperiosteum of the bilateral nasal septum during the septal deviation correction surgery, as well as the postoperative nasal cavity filling, and the formation of mucosal tissue scar, not only have a certain destructive effect on the nerve endings distributed in the nasal mucosa and affect the nerve conduction, but also destroy the nasal septal capillaries and glands, reduce the reactivity of the nasal mucosa, make it less sensitive to external physical and chemical adverse stimuli, and Inhibiting the sneezing reflex, reducing the secretion of active substances and decreasing the runny nose, all these factors may be beneficial in relieving the symptoms of allergic rhinitis.