The onset of cerebral hemorrhage is characterized by an acute onset, mostly during daytime exertion or emotional stress. Patients usually do not have any external abnormalities before onset, but may already have underlying diseases such as hypertension, venous malformations, and cerebral amyloid angiopathy. The most common cause of cerebral hemorrhage is hypertension combined with small arteriosclerosis, while other common causes include venous malformations, cerebral amyloid angiopathy, and blood disorders. Among them, long-term hypertension or cerebral amyloid angiopathy, which causes spontaneous rupture of small arteries, is about 85%, and is more likely to occur in middle-aged and elderly people over 50 years old, and people with hypertension, diabetes, hyperlipidemia and other underlying diseases are prone to bleeding under mood fluctuations, seasonal changes, staying up late, overworking, alcoholism and smoking. Most of the treatment for cerebral hemorrhage is based on internal medicine, quiet bed rest, and the use of drugs that lower intracranial pressure by dehydration, lower blood pressure, and prevent continued bleeding. If the condition is critical, such as high intracranial pressure and brain hernia formation, and if conservative medical treatment is not effective, surgical treatment is required in a timely manner. The prognosis of cerebral hemorrhage is related to the amount of bleeding, the site of bleeding, and the state of consciousness of the patient. Most patients have a poor prognosis, but some patients have a better recovery of neurological deficits.