Congenital biliary atresia is the most common condition that causes obstructive jaundice in newborns and requires surgical treatment. It is mainly classified according to the site of atresia as common bile duct atresia, hepatic duct atresia, and hilar hepatic duct atresia. Congenital biliary atresia is mostly in full-term infants and can be abnormal 1-2 weeks after birth, and the stool color is normal. 1. Jaundice: obstructive jaundice is the typical manifestation. Jaundice can be gradually obvious in 2-3 weeks after birth, or jaundice can be present after birth, which is often treated as physiological jaundice, after which the jaundice will gradually increase instead of receding. The sclera is brown, the skin changes from golden yellow to greenish-brown or dark green, and the stool may change gradually from yellow to pale yellow or clay. The urine may become thick tea-like as the jaundice deepens, and the diaper may become yellow. There is itching and scratching of the skin. Late stage cases show severe jaundice. 2, nutrition and stunting: Initially, the child is in good condition, with normal nutrition and development, and its performance does not correspond to the depth of jaundice. Later, the situation will gradually deteriorate, and by 3-4 months of age may be combined with fatty malnutrition and fat-soluble vitamin deficiency symptoms. 3. Hepatosplenomegaly: the liver is enlarged and hardened in texture. In the early stage, the spleen is not large and shows progressive enlargement as the disease progresses. Advanced cases may show abdominal distension, liver enlargement up to the umbilicus, and also accompanied by ascites and portal hypertension. Because untreated patients can die at 1-1.5 years of age and the disease can be normal in the early stages, it is important to diagnose and treat the disease promptly.