Cerebral infarction, also known as ischemic stroke, is a clinical syndrome in which the blood supply to the brain is impaired due to various causes, resulting in ischemic and hypoxic necrosis of the local brain tissue, followed by corresponding neurological deficits. According to the etiology, it is further divided into: 1. Large artery atherosclerosis type (cerebral thrombosis): infarction mainly due to atherosclerosis of large vessels, resulting in narrowing or occlusion of the vascular lumen. 2. Cardiogenic embolism type (cerebral embolism): its clinical manifestations and imaging are the same as those of large artery atherosclerosis type, but at least one cardiogenic stroke risk factor is highly or moderately present (e.g., the patient has a long-term history of atrial fibrillation, etc.). 3. Small artery occlusion type (lacunar cerebral infarction): it can have no obvious clinical manifestations or manifest as lacunar syndrome, and the infarct foci are usually less than 1.5 cm in diameter. 4. Other etiology type: it refers to infarcts due to rare etiologies other than the above three clear etiological typologies, such as blood composition changes and coagulation disorders. 5. Unknown etiology type: infarcts for which no etiology is found or cannot be typified.