Any cerebrovascular occlusion can lead to ischemia and infarction in the corresponding parts of brain tissue, and the most frequent infarction sites include cerebral cortex and subcortex, brainstem, cerebellum, internal capsule, and hemi-oval center. Infarcts caused by occlusion of the main trunk of the anterior or posterior circulation are larger, with severe symptoms and complex signs, mostly involving the cerebral cortex, brainstem, cerebellum and other parts of the brain, which can cause obvious symptoms such as impaired consciousness, aphasia, gaze, diplopia, central facial and tongue palsy, hemiparesis, hemianesthesia and ataxia. Patients are mostly left with obvious sequelae of neurological deficits, which affect the quality of life and have a poor prognosis. Deep penetrating branch lesions mostly cause lacunar cerebral infarction, which is commonly found in the brainstem, internal capsule, and hemi-oval center, etc. Patients have mild symptoms and single signs, and only have symptoms such as mild hemiparesis, sensory loss or abnormality, ataxia, aphasia, and cognitive function decline, and have a good prognosis.