The first thing we need to do is to confirm the diagnosis and find out if we have warts. 2, clinical performance: ① incubation period: 3 weeks to 8 months, an average of 3 months; ② symptoms and signs: male prepuce, glans, coronal groove, ties, penis, urethral opening, perianal and scrotum, etc., female for the labia, urethral opening, vaginal opening, perineum, perianal, vaginal wall, cervix, etc., passive anal sex can occur in the perianal, anal canal and rectum, oral sex can appear in the mouth. The lesions initially appear as small localized papules, the size of a pinhead to a green bean, gradually increasing in size or number, spreading to the surrounding area and developing into papillae, corns, cauliflower-shaped or masses. The lesions may be solitary or multiple. The color may range from pink to deep red (non-keratinized lesions), gray (severely keratinized lesions) or even brownish black (hyperpigmented lesions). A small number of patients develop large warts due to immunocompromise or pregnancy, which can involve the entire vulva, perineum, and gluteal sulcus, called giant warts. Patients usually have no conscious symptoms, but a few patients may feel itching, foreign body sensation, pressure or burning pain, and may experience rupture, maceration, erosion, bleeding or secondary infection due to increased brittleness of the lesions and friction. Female patients may have increased vaginal discharge. Subclinical and latent infections: subclinical infections have a normal surface appearance of the skin mucosa and may appear as well-defined whitish areas if a 5% acetic acid solution (white acetate test) is applied. Latent infection means that the tissue or cells contain HPV but the appearance of the skin mucosa is normal, the lesion proliferation keratinization is not obvious, and the acetic acid white test is negative. 3, laboratory tests: mainly histopathological examination and nucleic acid testing. ① Pathological examination: papilloma or warty hyperplasia, hyperkeratosis, lamellar hyperkeratosis, epidermal spine layer hypertrophy, basal cell hyperplasia, superficial dermal vascular dilatation, and lymphocyte-based inflammatory cell infiltration. Vacuolated cells in the superficial epidermis (granular layer and upper spiny layer) were seen in focal, lamellar and scattered distribution; sometimes densely stained granular material of varying sizes, i.e. viral inclusion bodies, could be seen in keratin-forming cells; ②Nucleic acid amplification test: amplification of HPV-specific genes (L1, E6, E7 region genes). A variety of nucleic acid detection methods are available, including fluorescent real-time PCR and nucleic acid probe hybridization tests. They should be carried out in laboratories accredited by relevant institutions. (B) Diagnostic criteria: 1, clinical diagnosis of cases: should be consistent with clinical manifestations, with or without epidemiological history.? 2, confirmed cases: should meet both the requirements of the clinical diagnosis of the case and laboratory tests in either one. Second, the relevant examination chapter Condyloma acuminatum before treatment usually need to do the relevant examination, mainly condyloma acuminatum confirmatory examination: white acetate test, image analysis, dermatoscopy, colposcopy, histopathological examination and HPV-DNA examination, the doctor will choose the appropriate examination according to the situation. The next is the routine examination before treatment: the four tests of infection, generally different hospitals will be different, and immunity test or have other necessary related tests. The actual fact is that you can find a lot of articles related to the treatment of condyloma acuminata. The actual fact is that you can find a lot of people who are not able to get a good deal on a lot of things. The actual fact is that you can find a lot of people who are not able to get a good deal on their own, and you need to review them regularly, usually every two weeks.