In general, facial neuritis (peripheral facial palsy) is characterized by asymmetrical frontal lines, poor ipsilateral eye closure or inability to close the eyes, low ipsilateral angle of the mouth, food retention between the cheeks and teeth, shallow ipsilateral nasolabial folds, salivation at the corners of the mouth, deviation of the corners of the mouth to the opposite side, and more pronounced deviation of the corners of the mouth to the opposite side by tooth movement (equivalent to grimacing). Simple (idiopathic) facial neuritis, also known as Bell’s palsy, occurs after cold, wind, flu, or prolonged depression and crying, and manifests as described above. It is common to have tearing in the ipsilateral eye. There is a kind of peripheral facial palsy caused by herpes zoster, called Hunt’s syndrome, in which the patient has pain in the affected auricle, external auditory canal, and mastoid area (i.e., the bone protrusion behind the ear), and herpes zoster appears a few days later (3-5 days) with the manifestation of peripheral facial palsy, and there may be loss of taste in the front 2/3 of the ipsilateral tongue. Early treatment can apply anti-inflammatory (hormonal drugs), anti-viral, neuroprotective and nutritional drugs, Chinese herbal medicine, physical therapy, corneal protection, and acupuncture after 1 week. Some patients may be left with sequelae, i.e., incomplete recovery, and some may develop ipsilateral facial muscle spasm. It should be differentiated from otogenic facial nerve palsy and peripheral facial palsy caused by parotitis, tumor compression, submandibular lymphadenitis, intracranial tumor compression, meningitis adhesions, etc. A doctor should be seen and relevant tests should be performed if necessary. The treatment of each disease should follow the principle of individualization, the above mentioned is only the direction or principle, for reference only, and the specific program gram depends on the specific situation of the patient. Herewith!