Traumatic brain injury is a common cause of hydrocephalus. Hydrocephalus is often secondary to severe craniocerebral injury. It is generally believed that hydrocephalus is caused by the accumulation of blood in the subarachnoid space and intracerebroventricular space after traumatic brain injury, blockage of the aqueduct and the outlet of the fourth ventricle by blood clots, which affects the circulation of cerebrospinal fluid and results in intracranial hypertension or obstruction of the subarachnoid villi by red blood cells and adhesions, preventing the absorption of cerebrospinal fluid. Therefore, both doctors and patients’ families should be alert to the occurrence of hydrocephalus after traumatic brain injury in patients. Post-traumatic hydrocephalus is divided into two categories: acute hydrocephalus occurs within two weeks after injury and chronic hydrocephalus occurs 3-6 weeks after injury. Acute hydrocephalus is mostly obstructive hydrocephalus, which is a pathological phenomenon caused by obstruction of the cerebrospinal fluid circulation pathway above the fourth ventricle, causing obstruction of the cerebrospinal fluid flow into the subarachnoid space (or cerebellar medullary pool). In simple terms, it is a blockage of a part of the fluid circulation system in our brain, resulting in an excessive accumulation of fluid in the brain, which affects brain function and produces the corresponding clinical symptoms. Chronic hydrocephalus is mostly traffic hydrocephalus, which is a pathological phenomenon caused by malabsorption or overproduction of cerebrospinal fluid and impaired excretion. Simply put, the fluid in the ventricles of the brain is not absorbed properly or is secreted excessively, resulting in more fluid coming in than going out and fluid accumulating in the brain. Regardless of the type of hydrocephalus mentioned above, all have broadly similar clinical manifestations, mainly headache, vomiting, apathy, unresponsiveness, and urinary incontinence. These can occur separately, but most often include more than two manifestations. The patient’s family needs to focus on the above symptoms during the accompanying process. Once they appear, respond to the doctor in time. Imaging is the most direct evidence of hydrocephalus. Enlargement of the ventricular system, rounding of the ventricular horns, thinning of the brain parenchyma, and paraventricular interstitial edema are characteristic manifestations of hydrocephalus. In summary, traumatic hydrocephalus is judged by: 1) a clear history of traumatic brain injury; 2) one or more of the above clinical manifestations; 3) imaging manifestations consistent with hydrocephalus. The above three aspects can be based on your own situation, and if you are not sure, you can consult the relevant professional doctors on the Internet to help judge. Experienced doctors can determine the type and severity of hydrocephalus based on the patient’s symptoms and imaging examinations. However, different patients have different conditions and need to be examined in detail and read films in detail, and sometimes need experimental treatment. Therefore, patients with hydrocephalus should be brought to the hospital in time to get more detailed and comprehensive information and to develop a targeted treatment plan.