Gout is a crystal-associated arthropathy caused by urate deposition, and its onset is directly related to hyperuricemia caused by disorders of purine metabolism and/or reduced uric acid excretion, which is a metabolic rheumatism. Gout is mainly caused by high blood uric acid levels in the body, and its onset is related to various factors such as long-term high purine diet, low water intake, and genetics. Gout is a metabolic rheumatism. When uric acid in the blood reaches a certain saturation state, urate crystals are precipitated and can be deposited in the distal joints, leading to acute joint inflammation and severe pain. Repeated attacks of acute joint inflammation can turn into chronic joint inflammation, and the continuous precipitation of urate crystals can lead to the formation of gout stones. Gout stones can appear in many parts of the body, causing damage to joints, cartilage, skin, and muscles. In addition, urate crystals can be deposited in the kidneys, causing kidney damage, such as gouty nephropathy or acute obstructive nephropathy. Patients with gout attacks must go to the hospital in a timely manner to improve systematic examination and active treatment to avoid further aggravation of the condition and organ damage. Gout patients are usually absolutely forbidden to drink alcohol because it is the main trigger for the occurrence of gout, and they should drink plenty of plain water every day to help the uric acid in the body to be excreted as soon as possible.