Gout is a recurrent inflammatory disease caused by increased purine biosynthesis, excessive uric acid production or poor uric acid excretion resulting in elevated uric acid in the blood and deposition of urate crystals in the synovial membrane, bursa, cartilage and other tissues of the joints. The disease is characterized by the presence of bifocal monohydrate urate crystals in joint fluid and gout stones. The clinical features are: hyperuricemia and characteristic acute arthritis due to urate crystals and deposition, gout stones, interstitial nephritis, and in severe cases, joint deformity and dysfunction, often accompanied by uric acid urinary tract stones. It is mostly seen in middle-aged and elderly men with obesity and postmenopausal women. With economic development and lifestyle changes, its prevalence is gradually increasing. It occurs in various parts of the body, with severe pain in the joints, and the pain is unbearable, but soon the pain blows over like “wind” in 1-7 days, so it is called “gout”. It is more common in men (95%) and in women after menopause because estrogen inhibits the formation of uric acid; however, the rate of attacks increases after menopause. There is no direct relationship between hyperuricemia and the occurrence of gout, only that hyperuricemia has a higher possibility of gout occurring. Some people with hyperuricemia will never trigger gout in their lifetime, while some people will have their first gout within a week or a month of discovering hyperuricemia. After the first gout, there is usually an interval of 1-2 years, and also a 10-year interval (5%), during which active treatment is needed to prevent the formation of gout stones. In our usual work, we found that in addition to active treatment of gout, it is more important to “nurture”. –The treatment of the acute stage (our specialty: internal and external treatment) Anti-inflammatory and analgesic drugs such as bupropion and fotarine are taken orally to control symptoms Sodium bicarbonate alkalinizes urine to promote uric acid excretion Drink 2000-2500 ml of water daily to accelerate uric acid excretion Betamethasone + lidocaine joint cavity injection to relieve symptoms Joint cold compresses to reduce swelling and relieve pain Low purine diet Myth of treatment in the acute stage: applying allopurine to lower uric acid may aggravate the symptoms again — Dr. Ling will give you tips on raising intermittent period 1. Take allopurine tablets to lower uric acid in the intermittent period and review uric acid regularly. 2, low purine diet: usually try to eat less of the following foods containing high purine such as animal offal, brain marrow, seafood, beef, mutton, ham, sausage, salted fish, chicken, goose, carp, rabbit, pigeon, lentils, peanut rice, bean food, etc. Recently there have been more cases of gout induced by eating hot pot. 3, eat less spicy and sour stimulating food: such as vinegar, prunes, wine, tea, coffee, spicy, etc. 4, moderate exercise such as brisk walking, jogging and swimming, weight control, maintain blood lipids, blood sugar stable in the normal range. 5, regular work and rest, do not stay up late. 6, drink more water, maintain the daily urine volume in more than 2000 ml.