Gout attacks are characterized by rapid onset, fast onset, and severe symptoms, mostly occurring at night and in single joints, with red, swollen, and hot joints that are painful. During the interval, people feel like normal people and do not feel anything unusual. However, it is not known that high blood uric acid level for a long time has an impact on many organs and tissues, especially on the kidneys and joint structures, and can cause a significant increase in the incidence of cardiovascular and cerebrovascular events. Because there are no obvious symptoms during the interval of gout, many patients are reluctant to take uric acid-lowering drugs, but when gout attacks and pain is unbearable, they think that gout caused by high blood uric acid should lower uric acid and eat uric acid-lowering drugs such as allopurinol and benzbromarone, which is extremely wrong! This is extremely wrong! I don’t know that the result of doing so makes uric acid in the tissues leach out and in turn aggravates the redness, swelling and heat of gout. Therefore, it is important to distinguish between the use of drugs for different periods of gout, treating the symptoms during attacks and the root cause during remission, and it is also wrong to take pain-relieving drugs only when the joints are swollen and painful. Colchicine, anti-inflammatory painkillers, non-steroidal anti-inflammatory drugs and even hormones can be applied to relieve pain quickly, but long-term uric acid-lowering treatment should be carried out in the interval to keep it at the ideal level for a long time, so as to avoid recurrent attacks of gouty arthritis from progressing to chronic arthritis and to prevent uric acid nephropathy and cardiovascular and cerebrovascular events.