Once amniotic fluid embolism occurs, the maternal mortality rate is very high, and some physicians even believe that almost all mothers will die if amniotic fluid embolism occurs. In most cases, the onset of amniotic fluid embolism is often preceded by prodromal symptoms such as chills, irritability, cough, shortness of breath, cyanosis, and vomiting. If the amount of amniotic fluid invasion is very small, the symptoms are mild and can sometimes recover on their own, while the typical clinical manifestations appear one after another when the amniotic fluid is cloudy or the amount is high. The following is the clinical manifestation of amniotic fluid embolism: 1, systemic bleeding tendency Some patients with amniotic fluid embolism are rescued to pass the period of respiratory and circulatory failure, followed by DIC, which is characterized by systemic bleeding tendency, such as skin, mucous membrane, bleeding from needle eyes and hematuria, etc., and the blood does not coagulate. However, some cases of amniotic fluid embolism lack respiratory and circulatory symptoms clinically, and the main manifestation of the disease is postpartum vaginal bleeding that cannot be easily controlled, which can be easily mistaken for postpartum bleeding caused by weak uterine contraction. 2.Respiratory and circulatory failure is divided into fulminant and slow type according to the condition. The fulminant form is followed by prodromal symptoms, followed by respiratory distress and cyanosis. In acute pulmonary edema, there is coughing, rapid heart rate, spitting of pink foamy sputum, and decrease or even disappearance of blood pressure. In a few cases, death occurs after cardiac arrest with only a scream. In the slow type, the respiratory and circulatory symptoms are mild or even absent, and are not diagnosed until the postpartum period when there is excessive bleeding and non-coagulation of blood. 3. Multi-system organ damage All organs of the body are damaged in this disease, except for the heart, the kidney is the most frequently damaged organ. As a result of kidney hypoxia, low urine, hematuria, urinary shutdown, azotemia, and death may occur due to renal failure; when the brain is hypoxic, patients may suffer from convulsions, irritability, and coma.