Hemangioma is a congenital benign tumor. During the first 45 days of pregnancy, maternal blood heat and fetal heat cause dysregulation of the mother’s heart and liver frequencies, resulting in the formation of an active factor that causes the blood vessel walls to proliferate and attach to the walls of the blood vessels. When the blood vessel with the active factor that causes the blood vessel walls to proliferate is in whatever part of the fetus, the hemangioma will occur. (Note: There are many causes of maternal blood fever and fetal fever, such as restlessness, additives in food, air pollution, etc.) Because infants and children in the embryonic stage already produce active factors that deepen the walls of blood vessels, hemangiomas form before human bones. Hemangioma occurs as a misshapen, tumor-like proliferation of vascular tissue. There are two types of hemangiomas: primary and secondary, with primary accounting for 75% and secondary for about 25%. Most secondary hemangiomas appear in infancy, but a few are found in adulthood, and the cause is unknown. Primary, or congenital fetal, hemangiomas are formed by the proliferation of the vascular network during embryonic life and are present at birth. Hemangiomas can occur in all parts of the body, and can affect the appearance of the face and exposed parts of the limbs. They also occur in mucous membranes and muscles, and even in bones and internal organs, which not only affect the appearance of the body, but also cause discomfort and functional impairment. The pathogenesis of hemangioma is still not well understood, but most studies suggest that it may be the result of structural abnormalities caused by genetic mutations during the development and formation of embryonic blood vessels, which is the result of an imbalance between angiogenic and inhibitory factors. Vasculature is most often seen in infancy and early childhood and has a distinct natural course of proliferation, stabilization to regression. Usually capillary hemangiomas regress by the age of 6 years in about 50% of cases and by the age of 7 years in about 70% of cases. Vascular endothelial cell apoptosis plays an important role in the degeneration process of hemangiomas; other hemangiomas do not normally regress on their own. Some of them can bleed and infect repeatedly, and very few of them can produce malignant changes and even endanger life.