Septic thrombosis is easily confused with the symptoms of the following conditions and differential diagnosis is noted. Traumatic thrombosis of the internal carotid artery The common carotid artery is the main arterial trunk of the head and neck, branching into the internal carotid artery and external carotid artery at the superior border of the thyroid cartilage. The incidence of traumatic embolism is higher in the internal carotid artery because the internal carotid artery mainly supplies blood to the brain and optic apparatus, so the consequences of embolism are more serious. Embolization of the internal carotid artery can occur in its cervical segment, petrous segment, cavernous sinus segment or supratentorial segment. According to the analysis of 70 cases, 85% of the cases occurred in the cervical segment and 10% in the petrous segment. Internal carotid artery embolism occurs most often after neck contusion patients may have transient cerebral ischemic attacks, followed by neurological signs, which are characteristic of internal carotid artery embolism. Venous thrombosis is a condition in which acute, nonsuppurative inflammation of the veins occurs with thrombosis under conditions of sluggish venous blood flow, hypercoagulability, and endothelial damage. The vast majority of venous thrombosis occurs in the deep veins of the pelvis and lower extremities. It is most common in patients after childbirth, fractures and trauma, and surgery. The possibility of this disease should be considered in the presence of limb pain, swelling, superficial vein varicose and palpable cords along the veins. Ultrasound Doppler and radionuclide venography can help in the diagnosis. Thrombosis is easily dislodged in the early stages and can cause large pulmonary infarcts, which are often a cause of sudden death. Thrombotic superfluous organisms superfluous organisms is a general term for all kinds of protrusions formed inside or outside the organism or organ in the course of pathology. Thrombotic redundancies are most commonly seen in non-bacterial thrombotic endocarditis. Non-bacterial thrombotic endocarditis can involve any heart valve, mainly affecting the mitral and aortic valves. Most of the redundancies in the mitral and tricuspid valves are located in the atrial plane, and most of the redundancies in the aortic and pulmonary valves are located in the ventricular plane. The main pathological change of nonbacterial thrombotic endocarditis is the formation of sterile organisms on the valves. Early changes are degenerative changes of the valve collagen and edema of the matrix under the effects of metabolic reactions, vitamin deficiencies, hemodynamic impairments, and aging, followed by localized stripping of the valve’s lining, which exposes the collagen and matrix to the blood stream. When the antibody is in a hypercoagulable state, platelets and other platelets can easily attach to its surface, forming nonbacterial thrombophilic endocarditis. Formation of non-bacterial thrombophilia. The lesions are superficial and often have no localized inflammatory reaction, and once detached, they can produce signs of arterial embolism.