Uric acid kidney stones are mainly caused by hyperuricemia, due to decreased solubility of uric acid in the urine or supersaturation of uric acid in the urine, which leads to the formation of urate crystals, which accumulate in the kidney and gradually increase, thus forming uric acid kidney stones. Uric acid kidney stones are generally diverse and fragile in shape, and the clinical differential diagnosis is made mainly by imaging features. x-ray plain examination shows translucent stones, so the main feature is that they do not show up on x-ray plain film. If uric acid kidney stones are mixed with calcium oxalate, calcium carbonate and other components, which in turn exhibit stone shadows of variable density, ultrasound reveals hyperechoic areas with acoustic shadows. The general density CT value of uric acid kidney stones is 300-400, which is much lower than other lesions. In addition, the urine of patients with uric acid kidney stones is mostly persistently acidic, i.e., the pH is mostly below 6.0, generally below 5.5, and the urine sediment examination shows light red urate crystals. If no urinary tract impaction and other conditions occur, there are mostly no obvious clinical symptoms, and if stone impaction occurs, it may lead to clinical manifestations such as back pain, hematuria and difficulty in urination. The main treatment for uric acid kidney stones is to lower uric acid, so that uric acid can be controlled within the ideal range, and urate stones can be gradually dissolved and made smaller, and then excreted from the body.