It is a spongy abnormal vascular mass composed of numerous sinusoidal dilated thin-walled vessels, not a tumor, and belongs to a type of vascular malformation according to histological classification and is an extra-axial lesion. The etiology is unknown and may be related to estrogen. It has been found that CSHs are more common in women, grow rapidly during pregnancy, and degenerate in the postpartum period, accounting for 2-3% of lesions in the cavernous sinus and 13% of intracranial cavernous hemangiomas. Prevalent in females aged 50-60 years, often solitary in individuals, no obvious familial pattern, higher incidence in Japanese population substantially malformed vascular masses. It does not contain true tumor tissue and is not a true tumor. To the naked eye, it is a purplish or dark red hemorrhagic mass with clear margins and a spongy or honeycomb-like cut surface, hence the name. Microscopically, there are abnormally dilated and congested blood sinuses, and the sinus walls are composed of monolayer endothelial cells and fibroblasts, lacking elastic fibers and muscle layers Imaging: üCT: well-defined borders, round, oval isointense or high-density shadow, mildly enhanced after enhancement, no surrounding edema, local bone destruction üMRI: high specificity. T1-weighted image with isosignal, T2-weighted image with high signal after enhancement, mixed signal shadow within the lesion. There is no circumferential low signal band. üDSA: arterial phase is rarely visualized, and late venous phase has dense venous pools and localized focal staining Microsurgery: Indication is significant occupational effect, severe or progressive focal neurological deficit. Removal of the lesion is prone to bleeding, leaving neurological dysfunction and difficult surgery. Temporal approach and frontotemporal approach can be used.