Spinal cord concussion is a state of transient functional inhibition following spinal cord injury. There are no obvious organic changes in the gross pathology, only a little edema under the microscope, and no destruction of nerve cells and nerve fibers is seen. The clinical manifestation is a delayed paralysis immediately after the injury below the plane of injury, and after a few hours to two days, the spinal cord function begins to recover without any neurological sequelae later. Spinal cord shock is a temporary and complete suppression of function that occurs when the spinal cord is severely traumatized and pathologically damaged, and is characterized by delayed paralysis, loss of all spinal cord reflexes, including pathological reflexes, and loss of diaphoresis. The systemic changes may include hypotension or decreased cardiac output, bradycardia, decreased body temperature, and respiratory dysfunction. Spinal shock occurs immediately after the injury. It usually lasts 3 to 4 days in children and 3 to 6 weeks in adults. The lower the site of spinal cord injury, the shorter the duration of its duration. For example, the period of spinal cord shock in the lumbar and sacral segments is usually less than 24 hours. The appearance of the bulbocavernosal reflex or anal reflex or plantar-plantar reflex is a marker for the end of spinal shock. After the end of the spinal shock period, if there is still no movement and sensation below the plane of injury, it indicates a complete spinal cord injury.