Facial palsy is the common name for facial neuritis, facial nerve paralysis. It is a common and frequent disease. Especially in winter and spring, the number of people suffering from facial palsy increases steeply due to climate change and cold wind stimulation. This is because when the weather becomes cold, the cold wind that blows directly on the face for a long time will cause spasm of the blood vessels that locally nourish the nerve due to over-cold stimulation, resulting in idiopathic (Bell’s palsy) due to ischemic edema of the nerve tissue. In fact, infection accounts for about 42.5% of the incidence in patients with facial palsy. Most of the common infectious lesions are caused by the activation of herpes zoster that is latent in the dormant state within the sensory ganglia of the facial nerve. In addition, meningitis, mumps, influenza, scarlet fever, malaria, polyneuritis, and localized infections can be caused. Bell’s palsy accounts for about 30.3% of the incidence. Tumors themselves and surgically removed tumors can cause Bell’s palsy, accounting for about 5.5% of the incidence. Common tumors include auditory neuroma, parotid adenoma, and primary cholesteatoma. Neurogenic facial palsy accounts for about 13.5% of the incidence and is caused by cerebrovascular disease, intracranial, and non-traumatic neurogenic causes. Temporal bone fracture, facial trauma, surgery and injection of neurotoxic drugs in the facial nerve distribution area are common traumatic causes of facial palsy, accounting for about 8.2% of the incidence. Symptoms of facial palsy: mainly expression muscle paralysis, drooping and skewing of the corners of the mouth to the healthy side, lacrimation or salivation, shallowing, disappearance or deepening of the nasolabial fold, or enlargement of the eye fissure, disappearance of the frontal line, or inability to frown, close the eyes, show the teeth, puff the cheeks, whistle, etc. If you suspect whether you are suffering from facial palsy, you can first perform self-examination, such as whether there is pain behind one ear or on the face. Whether the skin wrinkles on the forehead are the same, become lighter or disappear, and whether the outer part of the eyebrows and eyes are symmetrical and droopy. The size of the eye fissure, whether it is symmetrical, smaller or larger on both sides, whether the upper eyelid is drooping, whether the lower eyelid is ectropion, whether the eyelid is twitching and swollen, whether the conjunctiva is congested and ulcerated, whether there are symptoms of tearing, dryness, acidity and swelling. Whether there is tinnitus, stuffy ears, hearing loss. Whether the nasolabial folds become shallow, disappear or deepen. Whether the cheeks are symmetrical, flat, thickened or twitching. Whether the face feels tight, stiff, numb or atrophied. Whether the corners of the mouth are symmetrical, drooping, lifting or twitching; whether the lips of the mouth are swollen, and whether the human middle is oblique. Whether there is any change in the sense of taste. What’s more, check your facial activities, such as whether you can raise your eyebrows, frown, close your eyes, shrug your nose, show your teeth, nuzzle your mouth, and puff your cheeks. Modern medicine believes that the severity of facial palsy symptoms depends on the level of facial nerve damage and the degree of axonal degeneration, and the higher the level of facial nerve damage and the higher the degree of axonal degeneration, the more difficult it is to cure. For example, 15% of patients with Bell palsy may have severe neurodegeneration and destruction of the nerve lining, with ≥90% loss of facial nerve fibers on electronystagmography, with demyelination of the nerve as the main pathological change accompanied by axonal degeneration, and recovery of nerve function in patients with axonal degeneration depends on nerve regeneration, which can only be recovered after the nerve regeneration process is completed, usually taking several months to several years. It usually takes several months to years to recover, and often causes the appearance of several sequelae such as facial muscle spasm, linkage movement, and crocodile tears due to the misaligned growth of regenerated fibers. Facial palsy is a lesion of facial muscles and nerves at the same time. Only by grasping the opportunity and comprehensive treatment to repair both facial muscles and damaged facial nerves can we recover completely. Especially, early treatment is very important, which is directly related to the recovery of facial nerve and facial muscle function in later stages. For the different stages of peripheral facial palsy, the following treatment programs can be carried out based on the use of medication: high-frequency electrotherapy in the acute stage – to improve blood circulation and eliminate facial nerve edema; local medication – to control inflammation and activate nerve cells. Local medication in the recovery period – directly nourish and repair nerve cells; acupuncture with low-frequency electrical stimulation – stimulate facial muscle activity; facial massage – promote the coordination of facial muscle contraction; facial muscle function training – to restore the natural facial expression.