Chronic gastritis is the name of a disease in Western medicine, but in Chinese medicine it belongs to the category of gastric pain, fullness, poor appetite, noisy, belching, nausea, vomiting, and acidity. Chronic gastritis is a chronic inflammatory disease caused by damage to the gastric mucosa from a variety of different causes. If the gastric mucosa is congested and edematous, the mucosa is rough, the color is red and white, and in severe cases there is granular hyperplasia and erosion, the color is red and white with white predominating, the mucosa is thin, and the submucosal vascular network is permeable. Chronic gastritis is mainly characterized by its sporadic, recurring and long-lasting nature. Chronic gastritis includes chronic superficial gastritis and chronic atrophic gastritis, of which superficial and atrophic are divided into three grades: mild, moderate and severe. Chronic gastritis is a common disease, multi-morbidity, people often say “nine out of ten people stomach”, that is, nine out of ten people suffer from gastric disease. According to epidemiological surveys, the incidence of chronic gastritis in our large population is 76%. Hospital outpatient gastroscopy, 80-90% have varying degrees of inflammation of the gastric mucosa, indicating that chronic gastritis is indeed a common disease, the more frequent, and with the growth of age, the higher the incidence. The endoscopic typing of chronic gastritis is very complex and no unified opinion has been reached. Since the discovery of H. pylori (Hp) by Marshall in 1985, its pathogenicity in causing gastritis has been recognized worldwide. When chronic gastritis is done gastroscopically with biopsies taken from typical inflammatory sites in the stomach, atrophy of 1/3 of the gastric mucosal glands is mild atrophic gastritis, atrophy of 2/3 is moderate atrophic gastritis, and severe is most of the glandular atrophy. What is intestinal glandular epithelial metaplasia? The surface of the stomach is all gastric gland cells, which are columnar in shape, the same as above and below, and mainly secrete gastric acid, pepsin, and corrosive digested food. The intestinal gland epithelial cells are in the intestine, and the intestinal gland cells are cup-shaped, large at the top and small at the bottom. They absorb the nutrients from the food digested by the stomach on the surface of the intestine, and then transform them into refined substances through the ducts, and become angry and blood, and nourish all the limbs. If glandular cells of the intestine are found on the mucosal surface of the stomach, it is called “intestinal glandular epithelial hyperplasia”. What is the relationship between intestinal glandular epithelial hyperplasia and gastric cancer? Histopathological studies have revealed that gastric cancer is surrounded by intestinal glandular epithelial cells. Therefore, it is generally believed that gastric cancer arises on the basis of intestinal glandular epithelial hyperplasia, which is directly related to gastric cancer. Therefore, once intestinal glandular epithelial cells are found in the gastric mucosa, we should pay attention to treatment and strive to eradicate them to prevent cancer. What is atypical hyperplasia and is it cancerous? Atypical hyperplasia, also known as heterogeneous hyperplasia, can only be detected by histopathological section under high magnification microscope, and its morphology is a raised vesicle-like material on the side of the top of normal cells. Can chronic atrophic gastritis, intestinal glandular epithelial hyperplasia, and heterogeneous hyperplasia be cured? Does it recur after cure? At present, Western medicine considers atrophic gastritis irreversible, and when it is accompanied by intestinal glandular epithelial hyperplasia, or heterogeneous hyperplasia, surgery is often recommended to remove most of the stomach to prevent carcinogenesis, but after years of research in Chinese medicine, a large number of facts confirm that chronic atrophic gastritis can be cured and intestinal glandular epithelial hyperplasia and heterogeneous hyperplasia can disappear.