Alert for post-traumatic proptosis and intracranial murmurs

After traumatic brain injury, some patients will have protruding eyes and intracranial murmur, which is a typical manifestation of traumatic cavernous sinus fistula of the internal carotid artery, but some hospitals and doctors don’t have enough knowledge about this disease, which leads to some patients to ophthalmology and otorhinolaryngology, and even up to 7 years in our hospital, which causes great pain to the patient’s body and psychology, and even delays the treatment and increases the difficulty of the treatment, and sometimes causes blindness and other irreversible sequelae. Irretrievable sequelae. Internal carotid artery cavernous sinus fistula (CCF) refers to the intracranial cavernous sinus segment of the internal carotid artery itself or its branches in the cavernous sinus segment rupture, and the cavernous sinus to form an abnormal arterial and venous communication between the cavernous sinus, resulting in a series of clinical manifestations of the cavernous sinus and increased pressure in the cavernous sinus.CCF due to traumatic injuries caused by more than 75%, such as fracture tears of the base of the skull, bone fragments pierced, foreign body penetrating injuries, firearms injuries; other factors can occur Spontaneous CCF, such as aneurysm rupture, arteritis, atherosclerosis, spontaneous CCF during pregnancy. Typical clinical manifestations are as follows: 1, pulsatile protruding eyes (more than 95% of the literature), because of the increased pressure in the cavernous sinus, which affects the reflux of the ophthalmic vein; 2, tremor and murmur, which seriously affects the patient’s work and rest, this is the main reason for the patient’s visit to the clinic, so the process of treatment is based on the disappearance of the murmur as the standard; 3, tremor and murmur. The disappearance of murmur as the standard; 3, bulbar conjunctival edema and congestion, caused by the restriction of the return of the ophthalmic veins, is one of the reasons for the patient’s visit to the clinic; 4, limitation of ocular motility (uncommon), which is due to the compression of cranial nerves passing through the cavernous sinus; 5, vision loss; 6, neurological dysfunction and subarachnoid hemorrhage, which appear in the early stages of trauma, and are related to the location and degree of trauma; 7, fatal rhinorrhea, which is often associated with a pseudoaneurysm. Therapeutic aims of CCF: 1. to protect vision; 2. to eliminate murmurs; 3. to retract the eye; 4. to prevent cerebral ischemia or hemorrhage. Surgical treatment before the 1970s was roughly divided into 3 stages: Stage I (early 1800s-1930): ligation of the affected carotid artery, with an effective rate of 30%-40%, aggravated by ischemia and susceptible to recurrence; Stage II (1931-1960): isolation surgery. First ligation of the carotid artery, followed by craniotomy and clamping of the upper segment of the bed protrusion, with an effective rate of 56.9%; Stage III: kite tamponade. Craniotomy borrowed a puncture needle to introduce copper wire and cauda equina; individual reports of craniotomy for repair surgery under direct vision. However, due to the cumbersome craniotomy and the pain caused to the patient after leading to complications, as well as the difficulty in achieving the desired therapeutic effect or anatomical cure, so the hospitals with the conditions are now more common to use endovascular treatment. Since 1974, when Serbinenko first reported the successful treatment of TCCF with detachable balloon embolization, with the rapid development of medical imaging and the continuous improvement of embolization materials, as well as the continuous improvement of embolization techniques for more than 30 years, endovascular treatment with balloon embolization has become the preferred method of treatment of CCF. Sometimes, balloon embolization cannot be applied because the fistula is too small, and overlay stent or spring coil embolization treatment can be used. In cases where the fistula is too large to preserve the diseased vessel, the tumor-carrying artery can be occluded with good compensation.