The patient will feel uncomfortable in both lower extremities at night after going to bed or in a quiet state, and the discomfort is difficult to name, with a strong desire to move the legs and the need to get out of bed, which can seriously affect the patient’s sleep quality. The etiology and pathogenesis of restless legs syndrome are still unclear, but currently it is thought to be related to genetic factors, abnormal central dopamine function, abnormal iron metabolism and spinal cord conduction pathway lesions, etc. 1, genetic factors: according to clinical statistics, 50%-92% of patients with primary restless legs syndrome have a positive family history; 2, abnormal dopamine function: it is thought that abnormal central dopamine function may lead to restless legs syndrome; 3, iron deficiency or metabolic abnormalities: it is thought that abnormal central dopamine function may lead to restless legs syndrome. 3, iron deficiency or metabolic abnormalities: abnormal iron metabolism or reserve deficiency may be another important cause of restless legs syndrome, especially secondary restless legs syndrome; 4, spinal cord conduction pathway lesions: abnormalities in the spinal cord transmission pathway and spinal cord lesions may make the spinal cord superior neurons to the spinal cord issuing inhibitory impulses, resulting in increased sensitivity of the spinal cord flexor reflexes, thus causing restless legs syndrome.