Cerebral infarction, also known as ischemic stroke, is a softening necrosis of local brain tissue due to impaired blood circulation, ischemia and hypoxia. Cerebral infarction mainly develops due to atherosclerosis and thrombosis in the arteries supplying blood to the brain, causing luminal narrowing or even occlusion, resulting in focal acute cerebral blood supply deficiency, and also due to abnormal objects (solid, liquid, gas) entering the cerebral arteries along the blood circulation or the carotid arteries supplying cerebral blood circulation, causing blood flow blockage or sudden decrease in blood flow and producing softening necrosis of the brain tissue in the corresponding innervated area. The former is called arteriosclerotic thrombotic cerebral infarction, accounting for 40% to 60% of the disease, and the latter is called cerebral embolism accounting for 15% to 20% of the disease. In addition, there is still a kind of lacunar cerebral infarction, which is a microinfarction formed by occlusion of deep penetrating branches of cerebral arteries caused by hypertensive small arteriosclerosis, and it is also believed that a few cases can be caused by microembolism caused by disintegration of atherosclerotic plaque, and due to the popular application of CT and MRI, the incidence of which is quite high, accounting for about 20%-30% of cerebral infarction. Cerebral infarction is the most common cerebrovascular disease, accounting for about 75%, with an average death rate of 10%-15%, a very high disability rate, and a high risk of recurrence, and a significant increase in the mortality rate of recurrent stroke, which is one of the major diseases threatening human life and health today. About 25% of patients have a history of transient cerebral ischemic attack before the disease; young onset of cerebral arteritis of various causes is common; male onset is slightly more than female. Before the onset of the disease, there are mostly prodromal symptoms such as headache and dizziness, vertigo, transient limb numbness and weakness. The onset of the disease is generally slow, with most patients starting in silence and during sleep, and most patients reach their peak after a few hours or even 1 to 3 days. The neurological signs are mainly determined by the site of cerebrovascular occlusion and the extent of infarction, and are commonly manifested as focal neurological deficits such as aphasia, hemiparesis, and hemianesthesia. The disease is often accompanied by other complications, including lung infections, upper gastrointestinal bleeding, and decubitus ulcers. At this time, health education is particularly important. Take the 859 bed disease as an example, specific instructions are as follows: 1. 2, dietary guidance: instruct patients to consume low salt, low fat, low cholesterol food, such as animal offal, cream food. Choose foods that are easy to digest and high in vitamins. Change bad eating habits, each meal should not be too full, eat less and more meals, eat less spicy, too hot, too cold food, eat more fresh vegetables, fruits, cereals, fish and beans, so that the energy intake and needs to reach a balance. 3, medication guidance: oral medication should be taken on time and in the right amount. 4, daily life guidance: living environment to keep quiet and tidy, keep the history of the air fresh, appropriate temperature and moderate, avoid getting cold, soft light. Commonly used items in life are placed in places where patients can easily reach. The patient’s various excreta secretions should be handled in a timely manner. Assist in turning and patting the back regularly, turning every 2 hours to prevent the occurrence of pressure sores. Strengthen the functional exercise of the affected limbs and do a good job of skin cleaning care. 5. Knowledge guidance of the disease: guide the patient and family members to understand the basic causes, main risk factors and hazards of the disease, inform the early symptoms of the disease and the timing of consultation, master the knowledge of rehabilitation treatment and self-care methods of the disease, help analyze and eliminate factors that are not conducive to the recovery of the disease, and implement the rehabilitation plan. Both hemiplegia rehabilitation and language rehabilitation take a long time, have a high disability rate, and are prone to relapse. During the rehabilitation process, frequent contact with the rehabilitation physician should be made in order to adjust the treatment plan in time. Family members should care for the patient, give moral support and take care of the patient, avoid developing the patient’s dependency mentality, encourage and urge the patient to insist on exercise and enhance the ability of self-care. 6, examination guidance: CT examination is the most commonly used examination; MRI examination can show the size and location of ischemic tissue at an early stage. These two examinations require the removal of metal objects from the body. 7.Discharge instruction: ① Instruct the patient to do rehabilitation training every day, and the paralyzed limb should be kept in a good position. Massage the limb, do the passive training and gradually increase the amount of activity. When motor function starts to recover, patients should be encouraged to do early functional exercises of limbs and repulsion. ②Lower blood pressure and lipid. Antiplatelet therapy is the three cornerstones of cerebral infarction prevention and treatment. Patients should be instructed to take drugs on time and in quantity as prescribed by the doctor, and not to stop and reduce or change drugs without authorization. During the recovery period of cerebral infarction, pay attention to blood pressure control, and gradually control blood pressure below 140/90 mmHg. The antiplatelet drug aspirin should be taken for life, and the blood routine should be reviewed regularly. Long-term use of statins requires regular testing of liver enzymes and muscle enzymes. If liver enzymes exceed the upper limit of normal by 3 times and muscle enzymes exceed the upper limit of normal by 5 times, the drugs should be discontinued for observation. Taking anticoagulants requires regular check of platelets and coagulation function. ③Change the poor lifestyle, exercise properly, take reasonable rest and recreation, do not rely too much on family members in daily life, and try to do as much housework as you can, etc. Patients should move slowly when getting up, sitting or tying their shoes with their heads down, and should not turn their heads too hard or too fast. Pay attention to keeping warm when the climate changes to prevent colds. ④ Avoid recurrence factors: maintain emotional stability, avoid anger; eat in moderation, light and avoid greasy. Keep the bowels open, and if necessary, give open cork. ⑤When the patient has dizziness, headache, numbness and weakness of one limb, slurred speech or choking on food, fever, trauma, family members should promptly assist in medical consultation.