Patients with acute glomerulonephritis can develop edema, which can occur in 90% of patients with acute glomerulonephritis and is often the first cause of most patient visits. The cause of edema is due to reduced glomerular filtration rate and normal tubular reabsorption, resulting in a globular-tubular imbalance and thus water and sodium retention, leading to edema, increased blood volume, and elevated blood pressure. In addition, increased capillary permeability can further aggravate edema. Nephrogenic edema usually starts from the face, typically manifested as morning facial swelling or edema of both lower extremities, and in severe cases, pleural fluid, ascites and generalized edema may occur. Those with poor treatment results, especially those with combined acute pulmonary edema, need to undergo active dialysis treatment. Dialysis treatment is to help the patient to pass the dangerous period and return to normal when the kidney function is restored. Since the disease tends to heal spontaneously, maintenance dialysis therapy is generally not required. The prognosis of the acute phase of the disease is good, especially in pediatric patients, with the majority of patients having diuresis and decompensation, disappearance of sarcoid hematuria, and return to normal blood pressure within 2-4 weeks.