Facial palsy, commonly known as orofacial neuritis, is a common clinical disease characterized by sudden onset of facial expression muscle paralysis on one side, mainly manifesting as sudden onset, enlarged eye fissure on the affected side, inability to close the eyelid, tearing, loss of frontal lines, inability to frown, shallowing of the nasolabial folds on the affected side, and low skewing of the corners of the mouth to the healthy side. The exact cause of facial palsy is still unknown, but the main tendencies are: 1, acute viral or non-specific infections caused by immune or metabolic reactions, such as herpes, mumps, measles, influenza, cytomegalovirus and rheumatism; 2, lesions of the facial nerve itself, such as cold, trauma, poisoning and other factors attacking the blood vessels that nourish the facial nerve, resulting in local tissue ischemia, edema and compression of the facial nerve; 3, Peripheral factors resulting in facial nerve paralysis, such as lesions within the stem mammary foramen, compression of the facial nerve or impaired blood circulation operation, etc. At present, Western medical treatment in the acute phase is based on hormones and antiviral drugs. The sequelae period is mainly treated with dibazol, niacin and other vitamin B drugs, and the effect of simple western medical treatment is not very satisfactory.